Test bank for Rubin s pathology clinicopathologic foundations of medicine 7th edition by David S. Strayer, Emanuel Rubin Course

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RUBIN'SPATHOLOGY:CLINICOPATHOLOGICFOUNDATIONSOF MEDICINE
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7TH EDITION
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DAVIDS.STRAYER, EMANUELRUBIN
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,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
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Table of Contents:
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Chapter 1: Cell Adaptation, Injury and Death
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Chapter 2: Inflammation
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Chapter 3: Repair, Regeneration and Fibrosis
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Chapter 4: Immunopathology
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Chapter 5: Neoplasia
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Chapter 6: Developmental and Genetic Diseases
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Chapter 7: Hemodynamic Disorders
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Chapter 8: Environmental and Nutritional Pathology
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Chapter 9: Infectious and Parasitic Diseases
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Section II: Pathogenesis of Systemic Conditions Expandable section
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Chapter 10: Aging
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Chapter 11: Systemic Autoimmune Diseases
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Chapter 12: Sepsis
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Chapter 13: Obesity and Diabetes Mellitus
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Chapter 14: The Pathology of Pregnancy
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Chapter 15: The Amyloidoses
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Section III: Diseases of Individual Organ SystemsExpandable section
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Chapter 16: Blood Vessels
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Chapter 17: The Heart
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Chapter 18: The Respiratory System
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Chapter 19: The Gastrointestinal Tract
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Chapter 20: The Liver and Biliary System
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Chapter 21: The Pancreas
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Chapter 22: The Kidney
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Chapter 23: The Lower Urinary Tract and Male Reproductive System
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Chapter 24: The Female Reproductive System and Peritoneum
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Chapter 25: The Breast
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Chapter 26: Hematopathology
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Chapter 27: The Endocrine System
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Chapter 28: The Skin
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Chapter 29: The Head and Neck
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Chapter 30: Bones, Joints and Soft Tissue
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Chapter 31: Skeletal Muscle and Peripheral Nervous System
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Chapter 32: The Central Nervous System
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Chapter 33: The Eye
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Chapter 34: Forensic Pathology
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,Rubin's Pathology: Clinicopathologic Foundations of
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MedicineChapter 1: Cell Adaptation, Injury and Death
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Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a result
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1. of:
A) release of stored calcium from the mitochondria.g g g g g g




B) improved intracellular volume regulation. g g g




C) decreased influx across the cell membrane. g g g g g




D) attraction of calcium to fatty infiltrates. g g g g g




The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
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2. Adaptation to the reduced size of the liver leads to _
g ofthe remaining liver cells.
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A) metaplasia
B) organ atrophy g




C) compensatory hyperplasia g




D) physiologic hypertrophy g




A person eating peanuts starts choking and collapses. His airway obstruction is partially cleared,
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but he remains hypoxic until he reaches the hospital. The prolonged cell hypoxia
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3. caused a cerebral infarction and resulting
g g _ in the brain. g g g g g g




A) caspase activation g




B) coagulation necrosis g




C) rapidphagocytosis g




D) protein p53 deficiency g g




Bacteria and viruses cause cell damage by
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4. damage caused by other injurious agents.
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A) disrupting the sodium/potassium ATPase pump g g g g




B) interrupting oxidative metabolism processes g g g




C) replicating and producing continued injury g g g g




D) decreasing protein synthesis and function g g g g




The patient has a prolonged interruption in arterial blood flow to his left kidney, causing
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5. hypoxic cell injury and the release of free radicals. Free radicals damage cells by:
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A) destroying phospholipids in the cell membrane. g g g g g




B) altering the immune response of the cell. g g g g g g




C) disrupting calcium storage in the cell. g g g g g




D) inactivation ofenzymes and mitochondria. g g g g

, 6. Injured cells have impaired flow of substances through the cell membrane as a result of:
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A) increased fat load. g g




B) altered permeability. g




C) altered glucose utilization. g g




D) increased surface receptors. g g




7. Reversible adaptive intracellular responses are initiated by:
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A) stimulus overload. g




B) geneticmutations. g




C) chemical messengers. g




D) mitochondrial DNA. g




8. Injured cells become very swollen as a result of:
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A) increased cell protein synthesis. g g g




B) altered cell volume regulation. g g g




C) passive entry of potassium into the cell. g g g g g g




D) bleb formation in the plasma membrane.
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A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps on a
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piece of glass, the wound does not heal, and the area tissue becomes necrotic. The necrotic
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9. cell death is characterized by:
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A) rapid apoptosis. g




B) cellular rupture. g




C) shrinkage and collapse. g g




D) chronic inflammation. g




A 99-year-old woman has experienced the decline of cell function associated with age. A
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10. group of theories of cellular aging focus on programmed:
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A) changes with genetic influences. g g g




B) elimination of cell receptor sites. g g g g




C) insufficient telomerase enzyme. g g




D) DNAmutation or faulty repair.
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An 89-year-old female patient has experienced significant decreases in her mobility and stamina
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during a 3-week hospital stay for the treatment of a femoral head fracture. Which of the following
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phenomena most likely accounts for the patients decrease in muscle function
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11. thatunderlies her reduced mobility?
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A) Impaired muscle cell metabolism resulting from metaplasia g g g g g g




B) Dysplasia as a consequence of inflammation during bone remodeling g g g g g g g g




C) Disuse atrophy of muscle cells during a prolonged period of immobility
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