Terms in this set (94)
sustained
extended
(Meant to release the medication over a period of time)
enteric coded (delays the breakdown of the medication
Three types of coded until it gets past the stomach to protect it)
medication
how medication actually gains access to the body or enters
the bloodstream from the site of administration
Absorption
how the drug travels through the bloodstream to different
Distribution tissues in the body
metabolism how to body chemically modifies the drug
how the metabolized drug is removed from the body-
usually urine in the kidneys but can be through sweat,
excretion saliva, tears, and feces
, phenomenon of drug metabolism at a specific location in
the body which leads to a reduction in the concentration of
The first pass effect the active drug before it reaches the site of action.
metabolism
created to explain the active ingredient chemical structure,
action, or indication
Generic names of drugs
there is only one generic name
name given by the drug company.
brand names There are multiple brand names.
Group names for drugs that have similar activities or are
used for the same type of diseases and disorders.
usually mechanism of action (beta blockers, BPD 5
blockers), therapeutic classes (anti-hypertensives,
drug classes antiseizures)
their function and reassess dosing
what should you assess if a
patient has hepatic or renal
issues?
frequent urination, no urine formation
polyuria/anuria if the pt is chronic it is not a big deal but if it is a change it
is an emergency
, ibuprofen, naproxen, indomethacin, ketorolac, celecoxib
antipyretic/anti-analgesics
should only be on one of these medications.
nephrotoxic and hepatotoxic
MOA: cox 1 & 2 inhibitors
*except for celecoxib which is a cox 2 inhibitor. Cox 2
NSAIDS inhibits inflammation
Blackbox warning: GI bleeding
pt. complaining of GI upset can take an occult test.
, Bocks cox more centrally in the body.
antipyretic/anti-analgesics
NOT anti-inflammatory
preferred for some patients as it is NOT nephrotoxic but is
hepatotoxic. It can seriously damage the liver if pt
Acetaminophen overdoses on it.
N-acetylcysteine- used to treat acetaminophen toxicity.
hydrocortisone, prednisone, methylprednisolone,
dexamethasone
Best anti-inflammatory
Major systemic effects: hyperglycemia,
immunosuppression, hypertension, osteoporosis/fractures
preferential to select steroids with local effect versus a
Corticosteroids systemic effect such as: intranasal for COPD/allergies,
injection for gout, topically for rashes vs PO
sustained
extended
(Meant to release the medication over a period of time)
enteric coded (delays the breakdown of the medication
Three types of coded until it gets past the stomach to protect it)
medication
how medication actually gains access to the body or enters
the bloodstream from the site of administration
Absorption
how the drug travels through the bloodstream to different
Distribution tissues in the body
metabolism how to body chemically modifies the drug
how the metabolized drug is removed from the body-
usually urine in the kidneys but can be through sweat,
excretion saliva, tears, and feces
, phenomenon of drug metabolism at a specific location in
the body which leads to a reduction in the concentration of
The first pass effect the active drug before it reaches the site of action.
metabolism
created to explain the active ingredient chemical structure,
action, or indication
Generic names of drugs
there is only one generic name
name given by the drug company.
brand names There are multiple brand names.
Group names for drugs that have similar activities or are
used for the same type of diseases and disorders.
usually mechanism of action (beta blockers, BPD 5
blockers), therapeutic classes (anti-hypertensives,
drug classes antiseizures)
their function and reassess dosing
what should you assess if a
patient has hepatic or renal
issues?
frequent urination, no urine formation
polyuria/anuria if the pt is chronic it is not a big deal but if it is a change it
is an emergency
, ibuprofen, naproxen, indomethacin, ketorolac, celecoxib
antipyretic/anti-analgesics
should only be on one of these medications.
nephrotoxic and hepatotoxic
MOA: cox 1 & 2 inhibitors
*except for celecoxib which is a cox 2 inhibitor. Cox 2
NSAIDS inhibits inflammation
Blackbox warning: GI bleeding
pt. complaining of GI upset can take an occult test.
, Bocks cox more centrally in the body.
antipyretic/anti-analgesics
NOT anti-inflammatory
preferred for some patients as it is NOT nephrotoxic but is
hepatotoxic. It can seriously damage the liver if pt
Acetaminophen overdoses on it.
N-acetylcysteine- used to treat acetaminophen toxicity.
hydrocortisone, prednisone, methylprednisolone,
dexamethasone
Best anti-inflammatory
Major systemic effects: hyperglycemia,
immunosuppression, hypertension, osteoporosis/fractures
preferential to select steroids with local effect versus a
Corticosteroids systemic effect such as: intranasal for COPD/allergies,
injection for gout, topically for rashes vs PO
