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Summary Tutorial 3 Anxiety Disorders – Panic Disorder

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Relevant literature processed about panic disorder subjects with DSM criteria, with or without agoraphobia, hyperventilating, conditioning and treatment.

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May 3, 2016
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15
Written in
2014/2015
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Tutorial 3 Anxiety Disorders – Panic Disorder

What is a panic disorder?

Leerdoelen:
- What is a panic disorder according to DSM IV? Verschillen DSM V.
- What is agorafobie (in relatie tot paniek) according to DSM IV? Verschillen met DSM V.
- Epidemiologie
- Behandeling (exposure in vivo en CGT, verschillen en overeenkomsten)
- Is medicatie te adviseren?

Hyperventilatie
- Symptomen en fysiologische reactive
- Does it really help to breath into a sandwich bag.
- Hyperventilatiesyndroom and what is the hyperventilation provocation test.
- Do negative interpretations play a role in a panic attack?

Cognitieve model Clark 1986 en 1990. (Paniek cirkel)

INVULLEN MODEL MOWRER
Stage 1 – classical
Stage 2 – operant

CS UCS/UCR CR
Stage 1 Verhoogde hartslag Denken dat je een Zweten,
hartaanval krijgt hartkloppingen,
trillen
Sd R C
Stage 2 Verhoogde hartslag Frisse lucht (zie onder tabel)
opzoeken, in een zak
aderemn

Korte termijn
-C- (iets negatiefs wegnemen) – hartkloppingen nemen af
+C+ (iets positief toevoegen) – geruststelling geen hartaanval te krijgen
0C- (uitblijven catastrofe) – uitblijven hartaanval

Lange termijn
-C+ (afnemen iets positiefs) – niet meer leuke dingen kan doen omdat je bij een verhoging
van je hartslag meteen denkt een hartaanval te krijgen
+C- (iets negatiefs toevoegen) – angst in stand houden
0C+ (iets positiefs blijft uit) – nooit bevestiging dat je geen hartaanval krijgt omdat je het altijd
probeert te vermijden

COGNITIVE MODEL OF PANIC
Salkovskis & Beck
Hypothesis: a crucial component of the mechanism involved in acute panic attacks is the
catastrophic interpretation of bodily sensations. This interpretation results in further, anxiety-
induced bodily sensations, which augment the catastrophic interpretation, and so on round
in a vicious circle, culminating in a panic attack.

An implication of this model is that the vulnerability of particular patients to the experience of
acute panic (i.e. the basis of panic disorder as opposed to the experience of a single acute
panic attack) is not a direct result of biological pathology producing excessive autonomic
discharge, but rather is the result of a person’s enduring tendency to interpret bodily
R70,86
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