TEST BANK
RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF
MEDICINE 7TH EDITION
DAVID S. STRAYER, EMANUEL RUBIN
,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
sw sw sw sw sw sw sw sw sw
Table of Contents:
sw sw
Chapter 1: Cell Adaptation, Injury and Death
sw sw sw sw sw sw
Chapter 2: Inflammation
sw sw
Chapter 3: Repair, Regeneration and Fibrosis
sw sw sw sw sw
Chapter 4: Immunopathology
sw sw
Chapter 5: Neoplasia
sw sw
Chapter 6: Developmental and Genetic Diseases
sw sw sw sw sw
Chapter 7: Hemodynamic Disorders
sw sw sw
Chapter 8: Environmental and Nutritional Pathology
sw sw sw sw sw
Chapter 9: Infectious and Parasitic Diseases
sw sw sw sw sw
Section II: Pathogenesis of Systemic Conditions Expandable section
sw sw sw sw sw sw sw
Chapter 10: Aging
sw sw
Chapter 11: Systemic Autoimmune Diseases
sw sw sw sw
Chapter 12: Sepsis
sw sw
Chapter 13: Obesity and Diabetes Mellitus
sw sw sw sw sw
Chapter 14: The Pathology of Pregnancy
sw sw sw sw sw
Chapter 15: The Amyloidoses
sw sw sw
Section III: Diseases of Individual Organ SystemsExpandable section
sw sw sw sw sw sw sw
Chapter 16: Blood Vessels
sw sw sw
Chapter 17: The Heart
sw sw sw
Chapter 18: The Respiratory System
sw sw sw sw
Chapter 19: The Gastrointestinal Tract
sw sw sw sw
Chapter 20: The Liver and Biliary System
sw sw sw sw sw sw
Chapter 21: The Pancreas
sw sw sw
Chapter 22: The Kidney
sw sw sw
Chapter 23: The Lower Urinary Tract and Male Reproductive System
sw sw sw sw sw sw sw sw sw
Chapter 24: The Female Reproductive System and Peritoneum
sw sw sw sw sw sw sw
Chapter 25: The Breast
sw sw sw
Chapter 26: Hematopathology
sw sw
Chapter 27: The Endocrine System
sw sw sw sw
Chapter 28: The Skin
sw sw sw
Chapter 29: The Head and Neck
sw sw sw sw sw
Chapter 30: Bones, Joints and Soft Tissue
sw sw sw sw sw sw
Chapter 31: Skeletal Muscle and Peripheral Nervous System
sw sw sw sw sw sw sw
Chapter 32: The Central Nervous System
sw sw sw sw sw
Chapter 33: The Eye
sw sw sw
Chapter 34: Forensic Pathology
sw sw sw
,Rubin's Pathology: Clinicopathologic Foundations of
sw sw sw sw sw
MedicineChapter 1: Cell Adaptation, Injury and Deat
sw sw sw sw sw sw
h
Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a result
sw sw sw sw sw sw sw sw sw sw sw sw sw
1. of:
A) release of stored calcium from the mitochondria.
sw sw sw sw sw sw
B) improved intracellular volume regulation.
sw sw sw
C) decreased influx across the cell membrane.
sw sw sw sw sw
D) attraction of calcium to fatty infiltrates.
sw sw sw sw sw
The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw
2. Adaptation to the reduced size of the liver leads to _
sw sw sw of the remaining liver cells. sw sw sw sw sw sw sw sw sw sw sw
A) metaplasia
B) organ atrophy sw
C) compensatory hyperplasia sw
D) physiologic hypertrophy sw
A person eating peanuts starts choking and collapses. His airway obstruction is partially cl
sw sw sw sw sw sw sw sw sw sw sw sw sw
eared, but he remains hypoxic until he reaches the hospital. The prolonged cell hypoxia
sw sw sw sw sw sw sw sw sw sw sw sw sw
3. caused a cerebral infarction and resulting
sw sw _ in the brain.
sw sw sw sw sw sw
A) caspase activation sw
B) coagulation necrosis sw
C) rapid phagocytosis
sw
D) protein p53 deficiency sw sw
Bacteria and viruses cause cell damage by
sw sw sw sw sw sw , which is unique from the intracellular
sw sw sw sw sw sw
4. damage caused by other injurious agents.
sw sw sw sw sw
A) disrupting the sodium/potassium ATPase pump
sw sw sw sw
B) interrupting oxidative metabolism processes sw sw sw
C) replicating and producing continued injury sw sw sw sw
D) decreasing protein synthesis and function sw sw sw sw
The patient has a prolonged interruption in arterial blood flow to his left kidney, causing
sw sw sw sw sw sw sw sw sw sw sw sw sw sw
5. hypoxic cell injury and the release of free radicals. Free radicals damage cells by:
sw sw sw sw sw sw sw sw sw sw sw sw sw
A) destroying phospholipids in the cell membrane.
sw sw sw sw sw
B) altering the immune response of the cell.
sw sw sw sw sw sw
C) disrupting calcium storage in the cell.sw sw sw sw sw
D) inactivation of enzymes and mitochondria. sw sw sw sw
, 6. Injured cells have impaired flow of substances through the cell membrane as a result of:
sw sw sw sw sw sw sw sw sw sw sw sw sw sw
A) increased fat load. sw sw
B) altered permeability.sw
C) altered glucose utilization.
sw sw
D) increased surface receptors. sw sw
7. Reversible adaptive intracellular responses are initiated by:
sw sw sw sw sw sw
A) stimulus overload. sw
B) genetic mutations. sw
C) chemical messengers. sw
D) mitochondrial DNA. sw
8. Injured cells become very swollen as a result of:
sw sw sw sw sw sw sw sw
A) increased cell protein synthesis. sw sw sw
B) altered cell volume regulation.
sw sw sw
C) passive entry of potassium into the cell.
sw sw sw sw sw sw
D) bleb formation in the plasma membrane.
sw sw sw sw sw
A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps
sw sw sw sw sw sw sw sw sw sw sw sw sw s
won a piece of glass, the wound does not heal, and the area tissue becomes necrotic. The n
sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw
ecrotic
9. cell death is characterized by:
sw sw sw sw
A) rapid apoptosis.
sw
B) cellular rupture. sw
C) shrinkage and collapse. sw sw
D) chronic inflammation. sw
A 99-year-old woman has experienced the decline of cell function associated with age. A
sw sw sw sw sw sw sw sw sw sw sw sw sw
10. group of theories of cellular aging focus on programmed:
sw sw sw sw sw sw sw sw
A) changes with genetic influences.
sw sw sw
B) elimination of cell receptor sites. sw sw sw sw
C) insufficient telomerase enzyme. sw sw
D) DNA mutation or faulty repair.
sw sw sw sw
An 89-year-
sw
old female patient has experienced significant decreases in her mobility and stamina durin
sw sw sw sw sw sw sw sw sw sw sw sw
g a 3-
sw sw
week hospital stay for the treatment of a femoral head fracture. Which of the following ph
sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw
enomena most likely accounts for the patients decrease in muscle function
sw sw sw sw sw sw sw sw sw sw
11. that underlies her reduced mobility?
sw sw sw sw
A) Impaired muscle cell metabolism resulting from metaplasia
sw sw sw sw sw sw
B) Dysplasia as a consequence of inflammation during bone remodeling
sw sw sw sw sw sw sw sw
C) Disuse atrophy of muscle cells during a prolonged period of immobility
sw sw sw sw sw sw sw sw sw sw
RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF
MEDICINE 7TH EDITION
DAVID S. STRAYER, EMANUEL RUBIN
,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
sw sw sw sw sw sw sw sw sw
Table of Contents:
sw sw
Chapter 1: Cell Adaptation, Injury and Death
sw sw sw sw sw sw
Chapter 2: Inflammation
sw sw
Chapter 3: Repair, Regeneration and Fibrosis
sw sw sw sw sw
Chapter 4: Immunopathology
sw sw
Chapter 5: Neoplasia
sw sw
Chapter 6: Developmental and Genetic Diseases
sw sw sw sw sw
Chapter 7: Hemodynamic Disorders
sw sw sw
Chapter 8: Environmental and Nutritional Pathology
sw sw sw sw sw
Chapter 9: Infectious and Parasitic Diseases
sw sw sw sw sw
Section II: Pathogenesis of Systemic Conditions Expandable section
sw sw sw sw sw sw sw
Chapter 10: Aging
sw sw
Chapter 11: Systemic Autoimmune Diseases
sw sw sw sw
Chapter 12: Sepsis
sw sw
Chapter 13: Obesity and Diabetes Mellitus
sw sw sw sw sw
Chapter 14: The Pathology of Pregnancy
sw sw sw sw sw
Chapter 15: The Amyloidoses
sw sw sw
Section III: Diseases of Individual Organ SystemsExpandable section
sw sw sw sw sw sw sw
Chapter 16: Blood Vessels
sw sw sw
Chapter 17: The Heart
sw sw sw
Chapter 18: The Respiratory System
sw sw sw sw
Chapter 19: The Gastrointestinal Tract
sw sw sw sw
Chapter 20: The Liver and Biliary System
sw sw sw sw sw sw
Chapter 21: The Pancreas
sw sw sw
Chapter 22: The Kidney
sw sw sw
Chapter 23: The Lower Urinary Tract and Male Reproductive System
sw sw sw sw sw sw sw sw sw
Chapter 24: The Female Reproductive System and Peritoneum
sw sw sw sw sw sw sw
Chapter 25: The Breast
sw sw sw
Chapter 26: Hematopathology
sw sw
Chapter 27: The Endocrine System
sw sw sw sw
Chapter 28: The Skin
sw sw sw
Chapter 29: The Head and Neck
sw sw sw sw sw
Chapter 30: Bones, Joints and Soft Tissue
sw sw sw sw sw sw
Chapter 31: Skeletal Muscle and Peripheral Nervous System
sw sw sw sw sw sw sw
Chapter 32: The Central Nervous System
sw sw sw sw sw
Chapter 33: The Eye
sw sw sw
Chapter 34: Forensic Pathology
sw sw sw
,Rubin's Pathology: Clinicopathologic Foundations of
sw sw sw sw sw
MedicineChapter 1: Cell Adaptation, Injury and Deat
sw sw sw sw sw sw
h
Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a result
sw sw sw sw sw sw sw sw sw sw sw sw sw
1. of:
A) release of stored calcium from the mitochondria.
sw sw sw sw sw sw
B) improved intracellular volume regulation.
sw sw sw
C) decreased influx across the cell membrane.
sw sw sw sw sw
D) attraction of calcium to fatty infiltrates.
sw sw sw sw sw
The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw
2. Adaptation to the reduced size of the liver leads to _
sw sw sw of the remaining liver cells. sw sw sw sw sw sw sw sw sw sw sw
A) metaplasia
B) organ atrophy sw
C) compensatory hyperplasia sw
D) physiologic hypertrophy sw
A person eating peanuts starts choking and collapses. His airway obstruction is partially cl
sw sw sw sw sw sw sw sw sw sw sw sw sw
eared, but he remains hypoxic until he reaches the hospital. The prolonged cell hypoxia
sw sw sw sw sw sw sw sw sw sw sw sw sw
3. caused a cerebral infarction and resulting
sw sw _ in the brain.
sw sw sw sw sw sw
A) caspase activation sw
B) coagulation necrosis sw
C) rapid phagocytosis
sw
D) protein p53 deficiency sw sw
Bacteria and viruses cause cell damage by
sw sw sw sw sw sw , which is unique from the intracellular
sw sw sw sw sw sw
4. damage caused by other injurious agents.
sw sw sw sw sw
A) disrupting the sodium/potassium ATPase pump
sw sw sw sw
B) interrupting oxidative metabolism processes sw sw sw
C) replicating and producing continued injury sw sw sw sw
D) decreasing protein synthesis and function sw sw sw sw
The patient has a prolonged interruption in arterial blood flow to his left kidney, causing
sw sw sw sw sw sw sw sw sw sw sw sw sw sw
5. hypoxic cell injury and the release of free radicals. Free radicals damage cells by:
sw sw sw sw sw sw sw sw sw sw sw sw sw
A) destroying phospholipids in the cell membrane.
sw sw sw sw sw
B) altering the immune response of the cell.
sw sw sw sw sw sw
C) disrupting calcium storage in the cell.sw sw sw sw sw
D) inactivation of enzymes and mitochondria. sw sw sw sw
, 6. Injured cells have impaired flow of substances through the cell membrane as a result of:
sw sw sw sw sw sw sw sw sw sw sw sw sw sw
A) increased fat load. sw sw
B) altered permeability.sw
C) altered glucose utilization.
sw sw
D) increased surface receptors. sw sw
7. Reversible adaptive intracellular responses are initiated by:
sw sw sw sw sw sw
A) stimulus overload. sw
B) genetic mutations. sw
C) chemical messengers. sw
D) mitochondrial DNA. sw
8. Injured cells become very swollen as a result of:
sw sw sw sw sw sw sw sw
A) increased cell protein synthesis. sw sw sw
B) altered cell volume regulation.
sw sw sw
C) passive entry of potassium into the cell.
sw sw sw sw sw sw
D) bleb formation in the plasma membrane.
sw sw sw sw sw
A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps
sw sw sw sw sw sw sw sw sw sw sw sw sw s
won a piece of glass, the wound does not heal, and the area tissue becomes necrotic. The n
sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw
ecrotic
9. cell death is characterized by:
sw sw sw sw
A) rapid apoptosis.
sw
B) cellular rupture. sw
C) shrinkage and collapse. sw sw
D) chronic inflammation. sw
A 99-year-old woman has experienced the decline of cell function associated with age. A
sw sw sw sw sw sw sw sw sw sw sw sw sw
10. group of theories of cellular aging focus on programmed:
sw sw sw sw sw sw sw sw
A) changes with genetic influences.
sw sw sw
B) elimination of cell receptor sites. sw sw sw sw
C) insufficient telomerase enzyme. sw sw
D) DNA mutation or faulty repair.
sw sw sw sw
An 89-year-
sw
old female patient has experienced significant decreases in her mobility and stamina durin
sw sw sw sw sw sw sw sw sw sw sw sw
g a 3-
sw sw
week hospital stay for the treatment of a femoral head fracture. Which of the following ph
sw sw sw sw sw sw sw sw sw sw sw sw sw sw sw
enomena most likely accounts for the patients decrease in muscle function
sw sw sw sw sw sw sw sw sw sw
11. that underlies her reduced mobility?
sw sw sw sw
A) Impaired muscle cell metabolism resulting from metaplasia
sw sw sw sw sw sw
B) Dysplasia as a consequence of inflammation during bone remodeling
sw sw sw sw sw sw sw sw
C) Disuse atrophy of muscle cells during a prolonged period of immobility
sw sw sw sw sw sw sw sw sw sw
