Lippincott Illustrated Reviews
Pharmacology
Karen Whalen
8th Edition
,Lippincott Illustrated Reviews Pharmacology 8th Edition Karen Whalen TEST BANK
Table of Contents
UNIT I: Principles of Drug Therapy
Chapter 1: Pharmacokinetics
Chapter 2: Drug–Receptor Interactions and Pharmacodynamics
UNIT II: Drugs Affecting the Autonomic Nervous System
Chapter 3: The Autonomic Nervous System
Chapter 4: Cholinergic Agonists
Chapter 5: Cholinergic Antagonists
Chapter 6: Adrenergic Agonists
Chapter 7: Adrenergic Antagonists
Unit III: Drugs Affecting the Central Nervous System
Chapter 8: Drugs for Neurodegenerative Diseases
Chapter 9: Anxiolytic and Hypnotic Drugs
Chapter 10: Antidepressants
Chapter 11: Antipsychotic Drugs
Chapter 12: Drugs for Epilepsy
Chapter 13: Anesthetics
Chapter 14: Opioids
Chapter 15: Drugs of Abuse
Chapter 16: CNS Stimulants
UNIT IV: Drugs Affecting the Cardiovascular System
Chapter 17: Antihypertensives
Chapter 18: Diuretics
Chapter 19: Heart Failure
Chapter 20: Antiarrhythmics
Chapter 21: Antianginal Drugs
Chapter 22: Anticoagulants and Antiplatelet Agents
Chapter 23: Drugs for Hyperlipidemia
UNIT V: Drugs Affecting the Endocrine System
Chapter 24: Pituitary and Thyroid
Chapter 25: Drugs for Diabetes
Chapter 26: Estrogens and Androgens
Chapter 27: Adrenal Hormones
Chapter 28: Drugs for Obesity
UNIT VI: Drugs for Other Disorders
Chapter 29: Drugs for Disorders of the Respiratory System
Chapter 30: Antihistamines
Chapter 31: Gastrointestinal and Antiemetic Drugs
Chapter 32: Drugs for Urologic Disorders
Chapter 33: Drugs for Anemia
Chapter 34: Drugs for Dermatologic Disorders
Chapter 35: Drugs for Bone Disorders
Chapter 36: Anti-inflammatory, Antipyretic, and Analgesic Agents
UNIT VII: Chemotherapeutic Drugs
Chapter 37: Principles of Antimicrobial Therapy
Chapter 38: Cell Wall Inhibitors
Chapter 39: Protein Synthesis Inhibitors
Chapter 40: Quinolones, Folic Acid Antagonists, and Urinary Tract Antiseptics
Chapter 41: Antimycobacterial Drugs
Chapter 42: Antifungal Drugs
Chapter 43: Antiprotozoal Drugs
Chapter 44: Anthelmintic Drugs
Chapter 45: Antiviral Drugs
Chapter 46: Anticancer Drugs
Chapter 47: Immunosuppressants
UNIT VIII: Toxicology
Chapter 48: Clinical Toxicology
,Lippincott Illustrated Reviews Pharmacology 8th Edition Karen Whalen TEST BANK
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, Lippincott Illustrated Reviews Pharmacology 8th Edition Karen Whalen TEST BANK
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3. The nurse is teaching a patient who will be discharged home with a prescription for an enteric- coated
tablet. Which statement by the patient indicates understanding of the teaching?
a. I may crush the tablet and put it in applesauce to improve absorption.
b. I should consume acidic foods to enhance absorption of this medication.
c. I should expect a delay in onset of the drugs effects after taking the tablet.
d. I should take this medication with high-fat foods to improve its action. CORRECT ANSWER: C
Enteric-coated tablets resist disintegration in the acidic environment of the stomach and disintegrate when
they reach the small intestine. There is usually some delay in onset of actions after taking these
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medications. Enteric-coated tablets should not be crushed or chewed, which would alter the time and
location of absorption. Acidic foods will not enhance the absorption of the medication. The patient should
absorption rate.
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not to eat high-fat food before ingesting an enteric-coated tablet, because high-fat foods decrease the
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4. A patient who is newly diagnosed with type 1 diabetes mellitus asks why insulin must be given by
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subcutaneous injection instead of by mouth. The nurse will explain that this is because
a.
b.
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absorption is diminished by the first-pass effects in the liver.
absorption is faster when insulin is given subcutaneously.
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c. digestive enzymes in the gastrointestinal tract prevent absorption.
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d. the oral form is less predictable with more adverse effects. CORRECT ANSWER: C
Insulin, growth hormones, and other protein-based drugs are destroyed in the small intestine by digestive
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enzymes and must be given parenterally. Because insulin is destroyed by digestive enzymes, it would not
make it to the liver for metabolism with a first-pass effect. Subcutaneous tissue has fewer blood vessels,
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so absorption is slower in such tissue. Insulin is given subcutaneously because it is desirable to have it
absorb slowly.
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