test bank for rubins pathology clinicopathologic foundations of medicine 7th edition by david_s._strayer emanuel rubin.

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RUBIN'SPATHOLOGY:CLINICOPATHOLOGICFOUNDATIONSOF MEDICINE
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7TH EDITION
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DAVIDS.STRAYER, EMANUELRUBIN
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,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
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Table of Contents:
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Chapter 1: Cell Adaptation, Injury and Death
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Chapter 2: Inflammation
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Chapter 3: Repair, Regeneration and Fibrosis
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Chapter 4: Immunopathology
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Chapter 5: Neoplasia
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Chapter 6: Developmental and Genetic Diseases
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Chapter 7: Hemodynamic Disorders
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Chapter 8: Environmental and Nutritional Pathology
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Chapter 9: Infectious and Parasitic Diseases
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Section II: Pathogenesis of Systemic Conditions Expandable section
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Chapter 10: Aging
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Chapter 11: Systemic Autoimmune Diseases
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Chapter 12: Sepsis
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Chapter 13: Obesity and Diabetes Mellitus
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Chapter 14: The Pathology of Pregnancy
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Chapter 15: The Amyloidoses
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Section III: Diseases of Individual Organ SystemsExpandable section
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Chapter 16: Blood Vessels
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Chapter 17: The Heart
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Chapter 18: The Respiratory System
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Chapter 19: The Gastrointestinal Tract
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Chapter 20: The Liver and Biliary System
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Chapter 21: The Pancreas
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Chapter 22: The Kidney
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Chapter 23: The Lower Urinary Tract and Male Reproductive System
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Chapter 24: The Female Reproductive System and Peritoneum
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Chapter 25: The Breast
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Chapter 26: Hematopathology
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Chapter 27: The Endocrine System
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Chapter 28: The Skin
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Chapter 29: The Head and Neck
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Chapter 30: Bones, Joints and Soft Tissue
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Chapter 31: Skeletal Muscle and Peripheral Nervous System
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Chapter 32: The Central Nervous System
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Chapter 33: The Eye
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Chapter 34: Forensic Pathology
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,Rubin's Pathology: Clinicopathologic Foundations of
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MedicineChapter 1: Cell Adaptation, Injury and Death
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Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a result
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1. of:
A) release of stored calcium from the mitochondria.v v v v v v




B) improved intracellular volume regulation. v v v




C) decreased influx across the cell membrane. v v v v v




D) attraction of calcium to fatty infiltrates. v v v v v




The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
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2. Adaptation to the reduced size of the liver leads to _
v ofthe remaining liver cells.
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A) metaplasia
B) organ atrophy v




C) compensatory hyperplasia v




D) physiologic hypertrophy v




A person eating peanuts starts choking and collapses. His airway obstruction is partially cleared,
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but he remains hypoxic until he reaches the hospital. The prolonged cell hypoxia
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3. caused a cerebral infarction and resulting
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A) caspase activation v




B) coagulation necrosis v




C) rapid phagocytosis v




D) protein p53 deficiency v v




Bacteria and viruses cause cell damage by
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4. damage caused by other injurious agents.
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A) disrupting the sodium/potassium ATPase pump v v v v




B) interrupting oxidative metabolism processes v v v




C) replicating and producing continued injury v v v v




D) decreasing protein synthesis and function v v v v




The patient has a prolonged interruption in arterial blood flow to his left kidney, causing
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5. hypoxic cell injury and the release of free radicals. Free radicals damage cells by:
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A) destroying phospholipids in the cell membrane. v v v v v




B) altering the immune response of the cell. v v v v v v




C) disrupting calcium storage in the cell. v v v v v




D) inactivation of enzymes and mitochondria. v v v v

, 6. Injured cells have impaired flow of substances through the cell membrane as a result of:
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A) increased fat load. v v




B) altered permeability. v




C) altered glucose utilization. v v




D) increased surface receptors. v v




7. Reversible adaptive intracellular responses are initiated by:
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A) stimulus overload. v




B) genetic mutations. v




C) chemical messengers. v




D) mitochondrial DNA. v




8. Injured cells become very swollen as a result of:
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A) increased cell protein synthesis. v v v




B) altered cell volume regulation. v v v




C) passive entry of potassium into the cell. v v v v v v




D) bleb formation in the plasma membrane.
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A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps on a
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piece of glass, the wound does not heal, and the area tissue becomes necrotic. The necrotic
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9. cell death is characterized by:
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A) rapid apoptosis. v




B) cellular rupture. v




C) shrinkage and collapse. v v




D) chronic inflammation. v




A 99-year-old woman has experienced the decline of cell function associated with age. A
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10. group of theories of cellular aging focus on programmed:
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A) changes with genetic influences. v v v




B) elimination of cell receptor sites. v v v v




C) insufficient telomerase enzyme. v v




D) DNA mutation or faulty repair.
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An 89-year-old female patient has experienced significant decreases in her mobility and stamina
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during a 3-week hospital stay for the treatment of a femoral head fracture. Which of the following
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phenomena most likely accounts for the patients decrease in muscle function
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11. that underlies her reduced mobility?
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A) Impaired muscle cell metabolism resulting from metaplasia v v v v v v




B) Dysplasia as a consequence of inflammation during bone remodeling v v v v v v v v




C) Disuse atrophy of muscle cells during a prolonged period of immobility
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