TESTBANK N
RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF MEDICINE
N N N N N
7TH EDITION
N N
DAVID S. STRAYER, EMANUEL RUBIN
N N N N
,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
N N N N N N N N N
Table of Contents:
N N
Chapter 1: Cell Adaptation, Injury and Death
N N N N N N
Chapter 2: Inflammation
N N
Chapter 3: Repair, Regeneration and Fibrosis
N N N N N
Chapter 4: Immunopathology
N N
Chapter 5: Neoplasia
N N
Chapter 6: Developmental and Genetic Diseases
N N N N N
Chapter 7: Hemodynamic Disorders
N N N
Chapter 8: Environmental and Nutritional Pathology
N N N N N
Chapter 9: Infectious and Parasitic Diseases
N N N N N
Section II: Pathogenesis of Systemic Conditions Expandable section
N N N N N N N
Chapter 10: Aging
N N
Chapter 11: Systemic Autoimmune Diseases
N N N N
Chapter 12: Sepsis
N N
Chapter 13: Obesity and Diabetes Mellitus
N N N N N
Chapter 14: The Pathology of Pregnancy
N N N N N
Chapter 15: The Amyloidoses
N N N
Section III: Diseases of Individual Organ SystemsExpandable section
N N N N N N N
Chapter 16: Blood Vessels
N N N
Chapter 17: The Heart
N N N
Chapter 18: The Respiratory System
N N N N
Chapter 19: The Gastrointestinal Tract
N N N N
Chapter 20: The Liver and Biliary System
N N N N N N
Chapter 21: The Pancreas
N N N
Chapter 22: The Kidney
N N N
Chapter 23: The Lower Urinary Tract and Male Reproductive System
N N N N N N N N N
Chapter 24: The Female Reproductive System and Peritoneum
N N N N N N N
Chapter 25: The Breast
N N N
Chapter 26: Hematopathology
N N
Chapter 27: The Endocrine System
N N N N
Chapter 28: The Skin
N N N
Chapter 29: The Head and Neck
N N N N N
Chapter 30: Bones, Joints and Soft Tissue
N N N N N N
Chapter 31: Skeletal Muscle and Peripheral Nervous System
N N N N N N N
Chapter 32: The Central Nervous System
N N N N N
Chapter 33: The Eye
N N N
Chapter 34: Forensic Pathology
N N N
,Rubin's Pathology: Clinicopathologic Foundations of
N N N N
MedicineChapter 1: Cell Adaptation, Injury and Death
N N N N N N N
Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a result
N N N N N N N N N N N N N
1. of:
A) release of stored calcium from the mitochondria.
N N N N N N
B) improved intracellular volume regulation.
N N N
C) decreased influx across the cell membrane.
N N N N N
D) attraction of calcium to fatty infiltrates.
N N N N N
The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
N N N N N N N N N N N N N N N N N
2. Adaptation to the reduced size of the liver leads to _
N N N of the remaining liver cells. N N N N N N N N N N N
A) metaplasia
B) organ atrophy N
C) compensatory hyperplasia N
D) physiologic hypertrophy N
A person eating peanuts starts choking and collapses. His airway obstruction is partially
N N N N N N N N N N N N
cleared, but he remains hypoxic until he reaches the hospital. The prolonged cell hypoxia
N N N N N N N N N N N N N N
3. caused a cerebral infarction and resulting
N N _ in the brain.
N N N N N N
A) caspase activation N
B) coagulation necrosis N
C) rapid phagocytosis
N
D) protein p53 deficiency N N
Bacteria and viruses cause cell damage by
N N N N N N , which is unique from the intracellular
N N N N N N
4. damage caused by other injurious agents.
N N N N N
A) disrupting the sodium/potassium ATPase pump
N N N N
B) interrupting oxidative metabolism processes N N N
C) replicating and producing continued injury N N N N
D) decreasing protein synthesis and function N N N N
The patient has a prolonged interruption in arterial blood flow to his left kidney, causing
N N N N N N N N N N N N N N
5. hypoxic cell injury and the release of free radicals. Free radicals damage cells by:
N N N N N N N N N N N N N
A) destroying phospholipids in the cell membrane.
N N N N N
B) altering the immune response of the cell.
N N N N N N
C) disrupting calcium storage in the cell. N N N N N
D) inactivation of enzymes and mitochondria. N N N N
, 6. Injured cells have impaired flow of substances through the cell membrane as a result of:
N N N N N N N N N N N N N N
A) increased fat load. N N
B) altered permeability. N
C) altered glucose utilization.N N
D) increased surface receptors. N N
7. Reversible adaptive intracellular responses are initiated by:
N N N N N N
A) stimulus overload. N
B) genetic mutations. N
C) chemical messengers. N
D) mitochondrial DNA. N
8. Injured cells become very swollen as a result of:
N N N N N N N N
A) increased cell protein synthesis. N N N
B) altered cell volume regulation.
N N N
C) passive entry of potassium into the cell.
N N N N N N
D) bleb formation in the plasma membrane.
N N N N N
A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps on a
N N N N N N N N N N N N N N N
piece of glass, the wound does not heal, and the area tissue becomes necrotic. The necrotic
N N N N N N N N N N N N N N N N
9. cell death is characterized by:
N N N N
A) rapid apoptosis. N
B) cellular rupture. N
C) shrinkage and collapse. N N
D) chronic inflammation. N
A 99-year-old woman has experienced the decline of cell function associated with age. A
N N N N N N N N N N N N N
10. group of theories of cellular aging focus on programmed:
N N N N N N N N
A) changes with genetic influences. N N N
B) elimination of cell receptor sites. N N N N
C) insufficient telomerase enzyme. N N
D) DNA mutation or faulty repair.
N N N N
An 89-year-old female patient has experienced significant decreases in her mobility and
N N N N N N N N N N N
Nstamina during a 3-week hospital stay for the treatment of a femoral head fracture. Which of
N N N N N N N N N N N N N N N
Nthe following phenomena most likely accounts for the patients decrease in muscle function
N N N N N N N N N N N N
11. that underlies her reduced mobility?
N N N N
A) Impaired muscle cell metabolism resulting from metaplasia
N N N N N N
B) Dysplasia as a consequence of inflammation during bone remodeling
N N N N N N N N
C) Disuse atrophy of muscle cells during a prolonged period of immobility
N N N N N N N N N N
RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF MEDICINE
N N N N N
7TH EDITION
N N
DAVID S. STRAYER, EMANUEL RUBIN
N N N N
,Test Bank Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition
N N N N N N N N N
Table of Contents:
N N
Chapter 1: Cell Adaptation, Injury and Death
N N N N N N
Chapter 2: Inflammation
N N
Chapter 3: Repair, Regeneration and Fibrosis
N N N N N
Chapter 4: Immunopathology
N N
Chapter 5: Neoplasia
N N
Chapter 6: Developmental and Genetic Diseases
N N N N N
Chapter 7: Hemodynamic Disorders
N N N
Chapter 8: Environmental and Nutritional Pathology
N N N N N
Chapter 9: Infectious and Parasitic Diseases
N N N N N
Section II: Pathogenesis of Systemic Conditions Expandable section
N N N N N N N
Chapter 10: Aging
N N
Chapter 11: Systemic Autoimmune Diseases
N N N N
Chapter 12: Sepsis
N N
Chapter 13: Obesity and Diabetes Mellitus
N N N N N
Chapter 14: The Pathology of Pregnancy
N N N N N
Chapter 15: The Amyloidoses
N N N
Section III: Diseases of Individual Organ SystemsExpandable section
N N N N N N N
Chapter 16: Blood Vessels
N N N
Chapter 17: The Heart
N N N
Chapter 18: The Respiratory System
N N N N
Chapter 19: The Gastrointestinal Tract
N N N N
Chapter 20: The Liver and Biliary System
N N N N N N
Chapter 21: The Pancreas
N N N
Chapter 22: The Kidney
N N N
Chapter 23: The Lower Urinary Tract and Male Reproductive System
N N N N N N N N N
Chapter 24: The Female Reproductive System and Peritoneum
N N N N N N N
Chapter 25: The Breast
N N N
Chapter 26: Hematopathology
N N
Chapter 27: The Endocrine System
N N N N
Chapter 28: The Skin
N N N
Chapter 29: The Head and Neck
N N N N N
Chapter 30: Bones, Joints and Soft Tissue
N N N N N N
Chapter 31: Skeletal Muscle and Peripheral Nervous System
N N N N N N N
Chapter 32: The Central Nervous System
N N N N N
Chapter 33: The Eye
N N N
Chapter 34: Forensic Pathology
N N N
,Rubin's Pathology: Clinicopathologic Foundations of
N N N N
MedicineChapter 1: Cell Adaptation, Injury and Death
N N N N N N N
Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a result
N N N N N N N N N N N N N
1. of:
A) release of stored calcium from the mitochondria.
N N N N N N
B) improved intracellular volume regulation.
N N N
C) decreased influx across the cell membrane.
N N N N N
D) attraction of calcium to fatty infiltrates.
N N N N N
The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
N N N N N N N N N N N N N N N N N
2. Adaptation to the reduced size of the liver leads to _
N N N of the remaining liver cells. N N N N N N N N N N N
A) metaplasia
B) organ atrophy N
C) compensatory hyperplasia N
D) physiologic hypertrophy N
A person eating peanuts starts choking and collapses. His airway obstruction is partially
N N N N N N N N N N N N
cleared, but he remains hypoxic until he reaches the hospital. The prolonged cell hypoxia
N N N N N N N N N N N N N N
3. caused a cerebral infarction and resulting
N N _ in the brain.
N N N N N N
A) caspase activation N
B) coagulation necrosis N
C) rapid phagocytosis
N
D) protein p53 deficiency N N
Bacteria and viruses cause cell damage by
N N N N N N , which is unique from the intracellular
N N N N N N
4. damage caused by other injurious agents.
N N N N N
A) disrupting the sodium/potassium ATPase pump
N N N N
B) interrupting oxidative metabolism processes N N N
C) replicating and producing continued injury N N N N
D) decreasing protein synthesis and function N N N N
The patient has a prolonged interruption in arterial blood flow to his left kidney, causing
N N N N N N N N N N N N N N
5. hypoxic cell injury and the release of free radicals. Free radicals damage cells by:
N N N N N N N N N N N N N
A) destroying phospholipids in the cell membrane.
N N N N N
B) altering the immune response of the cell.
N N N N N N
C) disrupting calcium storage in the cell. N N N N N
D) inactivation of enzymes and mitochondria. N N N N
, 6. Injured cells have impaired flow of substances through the cell membrane as a result of:
N N N N N N N N N N N N N N
A) increased fat load. N N
B) altered permeability. N
C) altered glucose utilization.N N
D) increased surface receptors. N N
7. Reversible adaptive intracellular responses are initiated by:
N N N N N N
A) stimulus overload. N
B) genetic mutations. N
C) chemical messengers. N
D) mitochondrial DNA. N
8. Injured cells become very swollen as a result of:
N N N N N N N N
A) increased cell protein synthesis. N N N
B) altered cell volume regulation.
N N N
C) passive entry of potassium into the cell.
N N N N N N
D) bleb formation in the plasma membrane.
N N N N N
A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps on a
N N N N N N N N N N N N N N N
piece of glass, the wound does not heal, and the area tissue becomes necrotic. The necrotic
N N N N N N N N N N N N N N N N
9. cell death is characterized by:
N N N N
A) rapid apoptosis. N
B) cellular rupture. N
C) shrinkage and collapse. N N
D) chronic inflammation. N
A 99-year-old woman has experienced the decline of cell function associated with age. A
N N N N N N N N N N N N N
10. group of theories of cellular aging focus on programmed:
N N N N N N N N
A) changes with genetic influences. N N N
B) elimination of cell receptor sites. N N N N
C) insufficient telomerase enzyme. N N
D) DNA mutation or faulty repair.
N N N N
An 89-year-old female patient has experienced significant decreases in her mobility and
N N N N N N N N N N N
Nstamina during a 3-week hospital stay for the treatment of a femoral head fracture. Which of
N N N N N N N N N N N N N N N
Nthe following phenomena most likely accounts for the patients decrease in muscle function
N N N N N N N N N N N N
11. that underlies her reduced mobility?
N N N N
A) Impaired muscle cell metabolism resulting from metaplasia
N N N N N N
B) Dysplasia as a consequence of inflammation during bone remodeling
N N N N N N N N
C) Disuse atrophy of muscle cells during a prolonged period of immobility
N N N N N N N N N N
