Update) Adult Gerontology Management
Questions with Verified Answers | 100%
Correct| Graded A- UTA
What is hypokinesia?
I,- I,- I,-I,- I,- ➢ Hypokinesia—paucity of movement
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o Parkinson Disease [PD]
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o Parkinsonism or Parkinson-plus Syndromes—have some
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features of PD but also have other features; less or not responsive
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to dopaminergic drugs
I,- I,-
What is hyperkinesia?
I,- I,- I,-I,- I,- Hyperkinesia—too much movement I,- I,-
o Conditions that produce chorea—Huntington Disease;
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dyskinesias, dystonia, and tremor I,- I,- I,-
Tardive dyskinesia (TD) I,- a late-onset, irreversible neurologic I,- I,-I,- I,- I,- I,- I,- I,-
side effect of antipsychotic medications; characterized by
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abnormal, involuntary movements such as lip smacking, tongue
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protrusion, chewing, blinking, grimacing, and choreiform
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movements of the limbs and feet I,- I,- I,- I,- I,-
,Essential tremor a nerve disorder causing tremors to occur in
I,- I,-I,- I,- I,- I,- I,- I,- I,- I,- I,- I,-
a person who is moving or trying to move. Not usually associated
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with Parkinson's diseaseI,- I,-
Present when the limbs are in active use [purposeful or intention
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tremor]
Most common in the arms, but can affect—head, voice, legs
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Treatments for essential tremor Initial therapy—nonselective I,- I,- I,- I,-I,- I,- I,- I,-
β-blocker [propranolol, nadolol] or primidone [SOE=A] I,- I,- I,- I,- I,-
Other options—BaclofenOL, GabapentinOL, MirtazapineOL,
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PregabalinOL, and TopiramateOL have been described as I,- I,- I,- I,- I,- I,- I,-
effective in some patients, but results have not been consistent
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[SOE=B/C]
I,- OT can add additional benefit
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I,- Botulinum toxin may provide benefit is some cases I,- I,- I,- I,- I,- I,- I,-
Severe, medically refractory tremor may be treated with deep
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brain stimulation I,-
Pernicious anemia I,- I,-I,- I,- lack of intrinsic factor, decreased b12
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Pernicious anemia s/s I,- I,- I,-I,- I,- beefy red tongue, fatigue, paresthesia
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of hands/feet, fatigue
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,macrocytic anemias I,- I,-I,- I,- Pernicious anemia, folate deficiency I,- I,- I,- I,-
anemia >103mm I,-
causes of macrocytic anemias
I,- I,-liver disease, low b12 or folate, I,- I,-I,- I,- I,- I,- I,- I,- I,- I,-
gastrectomy, malabsorption, alcoholics I,- I,-
microcytic anemia iron deficiency, anemia of chronic disease
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late stage, lead poisoning, thalassemias,, occult blood in stool,
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menorrhagia, <87mm I,-
normocytic anemia anemia of chronic disease, sickle cell,
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impaired bone marrow, hemolytic anemia, 87-103mm
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primary storage for iron
I,- I,- I,- I,-I,- I,- ferritin >100 = normal I,- I,- I,-
increased ferritin I,- I,-I,- I,- inflammatory disease, hepatitis, CRF, I,- I,- I,-
transferrin regulates iron absorption and transport in body,
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low levels = protein malnutrition, >200 is normal
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total iron binding capacity (TIBC)
I,- I,- I,- I,- I,-I,- I,- high when iron low 240-450
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, > 400 with iron deficiency anemia
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Primary hypothyroidism I,- I,-I,- I,- High TSH, low T3 and T4
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Secondary hypothyroidism I,- I,-I,- I,- low TSH, low T3/T4, malfunction
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of pituitary
I,-
Hashimoto's thyroiditis I,- I,-I,- I,- autoimmune thyroiditis, high TSH, I,- I,- I,- I,-
low T3/T4I,-
Myxedema I,-I,- I,- puffy face with hypothyroidI,- I,- I,-
Grave's disease autoimmune disorder leading to
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hyperthyroidism, antibodies mimicking TSH, high T4/T3 I,- I,- I,- I,- I,-
Primary Hyperthyroidism I,- I,-I,- I,- Low TSH High T3,T4
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excessive iodine uptake I,- I,- I,-I,- I,- hyperthyroidism
first test for thyroid
I,- I,- I,- I,-I,- I,- TSH
TSH improvement after levothyroxine
I,- I,- I,- I,-I,- I,- 6-8 weeks I,-