RELIAS Fetal Heart Monitoring Exam 2025 –
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uterine blood supply
- uterine arteries deliver oxygenated blood to spiral arteries which
bring oxygen rich blood to intervillous space of placenta that has
fetal capillaries
- fetal capillaries carry the O2 rich blood to umbilical VEIN that
goes to fetus
-in contrast, the umbilical ARTERIES return waste products to that
intervillous space that go into mother's venous system
Potential issues that negatively affect fetal oxygenation Maternal
Oxygenation: asthma, hyper- or hypo- ventilation
Maternal Circulation: decreased maternal cardiac output, hypotension,
decreased Hgb
Placental O2 and CO2 Exchange: postterm, abruption, HTN,
hypotension, uterine tachysystole
Fetal circulation: cord compression or occlusion
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Fetal hypoxemia
- can occur d/t reduced fetal O2 reserves, excessive uterine activity,
or reduced uteroplacental blood flow
- worsening fetal hypoxemia can lead to abnormal FHR patterns,
mostly minimal or absent variability from acidemia
(1) hypoxemia vs. (2) hypoxia
1 - reduce O2 in blood
2 - reduced O2 delivery at tissue level
Fetal anaerobic metabolism
- occurs when long term O2 delivery is insufficient to meet cellular
needs of tissues
- results in production of lactic acid and other noncarbonic acids
- ACIDOSIS is the presence of excessive acids in tissues
acidosis pH below
7.35 pH is low
(acidosis is the process that leads to low blood pH, or acidemia)