MARYVILLE NURS 615 EXAM 3: 50 QUESTIONS AND 100%
VERIFIED ANSWERS 2025 /GRADED A+C/GUARANTEED PASS!!
1st line diuretic treatment for heart failure?
- answer--Spironalactone or furosemide?
Black box warning for pioglitazone?
- answer--ischemic cardiovascular risk and heart failure
Contraindications of acarbose?
- answer-Contraindicated w/ inflammatory or irritable bowel syndrome
Contraindications w/ ARBs?
- answer-Pregnancy
Contraindications w/ Digoxin and NSAIDs?
- answer-give acetaminophen for pain
Education needed for alpha-glucosidase inhibitors?
- answer-Must be taken w/ first bite of food from each meal
Education needed with long-term corticosteroid use?
- answer--Never stop abruptly
-take prilosec or zantac to protect from GI bleeds
-Diabetics may need more insulin
-Monitor for signs of infection
First line treatment for Acute gout attack?
- answer-colchicine
First line treatment for mild to moderate pain?
- answer-Ibuprofen and tylenol
How long is NPH onset of action?
- answer---1 hour onset
, Initial dose for 70 yo female w/ HTN and giving CCB
- answer-?
Low dose colchicine versus high dose colchicine?
- answer-low: 1.2 mg initially, 0.6 mg one hour later
High: 0.6-1.2mg every hour until relief. Max 4-8mg
MOA of alpha-glucosidase inhibitors (acarbose and miglitol)
- answer-inhibits absorption of carbs thus decreasing amount of glucose available
for absorption
MOA of CCB?
- answer--Prevents the opening of calcium channels, reducing the effect of calcium
excitation, contraction, coupling of skeletal, smooth and cardiac muscle, regulating
aldosterone and pacemaker signal conduction
= decreased contraction strength
MOa of cholestyramine (questran)?
- answer-exchange chloride ions for bile acids promoting an increase in bile acid
secretion
MOA of diltazem?
- answer-Same as CCB, Most likely to affect myocardial contractility and AV
conduction
MOa of dipeptidyl peptidase-4 inhibitors (gliptins)
- answer-Block DPPD-4 by increasing incretin levels, extends action of GLP-1 and
GIP to inhibit glucagon release which increases insulin secretion; decreases gastric
emptying and blood glucose
MOA of ibuprofen?
- answer-inhibit COX enzymes (cyclooxygenase)
-prostaglandin synthetase
-Inhibition of Cox 1 and 2 pathways decreases prostaglandin formation that causes
pain response and inflammation
MOA of metformin?
- answer-Stimulates infulin release from pancreatic beta cells to decrease blood
glucose
VERIFIED ANSWERS 2025 /GRADED A+C/GUARANTEED PASS!!
1st line diuretic treatment for heart failure?
- answer--Spironalactone or furosemide?
Black box warning for pioglitazone?
- answer--ischemic cardiovascular risk and heart failure
Contraindications of acarbose?
- answer-Contraindicated w/ inflammatory or irritable bowel syndrome
Contraindications w/ ARBs?
- answer-Pregnancy
Contraindications w/ Digoxin and NSAIDs?
- answer-give acetaminophen for pain
Education needed for alpha-glucosidase inhibitors?
- answer-Must be taken w/ first bite of food from each meal
Education needed with long-term corticosteroid use?
- answer--Never stop abruptly
-take prilosec or zantac to protect from GI bleeds
-Diabetics may need more insulin
-Monitor for signs of infection
First line treatment for Acute gout attack?
- answer-colchicine
First line treatment for mild to moderate pain?
- answer-Ibuprofen and tylenol
How long is NPH onset of action?
- answer---1 hour onset
, Initial dose for 70 yo female w/ HTN and giving CCB
- answer-?
Low dose colchicine versus high dose colchicine?
- answer-low: 1.2 mg initially, 0.6 mg one hour later
High: 0.6-1.2mg every hour until relief. Max 4-8mg
MOA of alpha-glucosidase inhibitors (acarbose and miglitol)
- answer-inhibits absorption of carbs thus decreasing amount of glucose available
for absorption
MOA of CCB?
- answer--Prevents the opening of calcium channels, reducing the effect of calcium
excitation, contraction, coupling of skeletal, smooth and cardiac muscle, regulating
aldosterone and pacemaker signal conduction
= decreased contraction strength
MOa of cholestyramine (questran)?
- answer-exchange chloride ions for bile acids promoting an increase in bile acid
secretion
MOA of diltazem?
- answer-Same as CCB, Most likely to affect myocardial contractility and AV
conduction
MOa of dipeptidyl peptidase-4 inhibitors (gliptins)
- answer-Block DPPD-4 by increasing incretin levels, extends action of GLP-1 and
GIP to inhibit glucagon release which increases insulin secretion; decreases gastric
emptying and blood glucose
MOA of ibuprofen?
- answer-inhibit COX enzymes (cyclooxygenase)
-prostaglandin synthetase
-Inhibition of Cox 1 and 2 pathways decreases prostaglandin formation that causes
pain response and inflammation
MOA of metformin?
- answer-Stimulates infulin release from pancreatic beta cells to decrease blood
glucose
