WGU D027 Exam/ New WGU D027 Actual Exam/ All
m m m m m m m m
Questions with Correct Answers/ Updated
m m m m m
2024-2025.
Whichmof mthe mfollowingmismnotmamcellularmfunction?m-mANSWERm-mCombustionm-
Allmlivingmcellsmmustmabsorbmandmmetabolizemsourcesmofmenergy,mexcretemwastemandmby-
productsmofmthose mprocesses,mandmreproducemtomcontinue moffspring.mPyrotechnicmprocessesm(i.e.mcom
bustion) mare mgenerallymmetaphorical.
Whatmare mmitochondriamresponsible mfor?m-mANSWERm-mEnergymproductionm-
Mitochondriamare mthemenergymproductionmorganellesmof mthe mcellmresponsible mformthemmajormmetabolicmp
rocessesmofmcreatingmunitsmofmenergym(e.g.,mATP).
Whichmifmthe mfollowingmcanmcause medema?m-mANSWERm-mIncreasedmlymphaticmpressure m-
Decreasedmvascularmandmlymphaticmpressuremdoesmnotmgenerallymresultminmthemmovementmandmaccumula
tionmofmfluidmintomextravascularmspaces.mIncreasedmplasmampressureminmthe mvascularmresultsminmhyperten
sion,
while mincreasedmlymphaticmpressuremwillmreadilymresultminmthempoolingmof mlymphaticmdrainage mintomthirdm
spacesm(i.e.medema).
Whichmof mthe mfollowingmbestmdescribesmthe mresultmof mcellularmreproduction?m-mANSWERm-
m Twom diploid m cells, m calledm daughterm cells, m are m produced m -
The mresultantmoffspringmofmcellularmreproductionmismthemcreationmofmtwomdaughtermcellsmaftermthemcomple
tionmofmmitosismormmeiosis.mInterphasemismthe mlongestmperiodmthatmamcellmspendsmitsmlifemcycle mandmismth
e
preparatorymstage mbeforemcellularmreproduction.mAnaphasemismthemseparationmofmgeneticmmaterialsmtomthemop
posingmcentromeresmof mthe mcell,mfollowedmbymtelophase.
Whatmindicatesmhypokalemia?m-mANSWERm-mSerummKmdecreasesmtomlessmthanm3.5m-
pg.m
1
,Normalmserummpotassiumm(K),malsomknownmasmeukalemia,mismgenerallym3.5-
5.5mmEq/L,mwithmsome mvariationsmof m+/-m0.1-
0.3mmEq/Lmdependingmonmthe mlaboratorymtesting,mage,mandmothermpotentialmpatient-
specificmconditionsmandmvariations.mAlterationsminmfluidmvolumemormthemadministrationmofmamsupplementm
alone mdomnotmnecessarilymcontribute mtomthe mdevelopmentmof mhypokalemia.
Whatmismanmexample mof mbothmhyperplasiamandmhypertrophy?m-mANSWERm-
m Uterine m enlargementm due m tom pregnancy m -
Hyperplasiamismthe mincrease minmthe mnumbermofmcellsmof manmorganmormtissue,mwhile mhypertrophymismthe
enlargementmofmthe mcellsmofmamgivenmorganmormtissuemwithoutmamchangeminmthe mnumbermofmcells.mMuscle
m cellsm grow m andm enlargem (hypertrophy)minm responsemtomincreased mwork m orm stressm frommexercise m orm incr
eased
cardiovascularmpressures.mWhenmamlivermismdamagedmormpartmofmitmismremoved,mitmismthe mone minternalmsol
idmorganmcapable mofmregenerationmtomrepairmitselfmcreatingmmoremcellsm(hyperplasia).mDuringmpregnancy,m
the muterusmmustmbothmgrowmtomcreatemmore mcellsmandmenlarge mthosemexistingmcellsminmpreparationmformt
he msupportmof mamfetus.
Whatmregulatesmthe msodiummbalance?m- mANSWERm-mAldosterone m-
Aldosterone mismamregulatorymhormone mproducedmbymthemadrenalmglandsmtomaffectmthemkidneys'mregulationmof
m sodium, m potassium, m andm waterm excretion.
Whatmismthe malterationmifmthe mextracellularmfluidmvolume mismlessmthanmnormal?m-mANSWERm-mHypotonicm-
Inmhypotonicmstates,mthemcellmsizemismincreasedmasmamresultmofmwatermmovingmfrommthemextracellularmfluidm
(ECF) mintomthe mcellmtomincrease mintracellularmfluid.mIso-
m orm normotonicm states m are m am balanced m state m between m intra-
m andm extracellularm fluidmvolumes. mHypertonicm statesm cause mwatermtomshiftmoutmofm them cellm tom increase m t
he mECFmabove mnormal.
Whenminmexcess,mwhatmdombuffersmabsorb?m-mANSWERm-mHydrogenm-
Excessesmofmacidmormbase,mH+ mormhydroxylmionm(OH−),mare mabsorbedmformthempurpose mof mmaintainingmpHmho
meostasism(pHmof m7.38-7.42)
Whichmof mthe mfollowingmwouldmampatientmwithmmetabolicmacidosismhave?m-mANSWERm-
m pHm below m 7.35;m Bicarbonate m levelm below m 22m mEq/Lm -
The mdiagnosismofmmetabolicmacidosismrequiresmthatmthe mtestingmof marterialmbloodmfindsmthatmthe mpHmisml
essmthanm7.35mandmthe mbicarbonate mlevelmismlessmthanm22mmEq/L.
pg.m
2
,Whatmismthe mprocessmbymwhichmRNAmdirectsmthe msynthesismofmpolypeptides?m-mANSWERm-mTranslationm-
pg.m
3
, Translationmrefersmtomthismprocess.mTranscriptionmismanothermprocessminmwhichmRNAmismsynthesized.mMut
ationmmeansmgeneticmmaterialmhasmbeenmmodifiedmthroughminheritance.mCreationmismamtermmthatmdoesmn
otmapplyminmthismcontext.
Whatmismthe mrecurrence mrate mformautosomalmdominantmdisease?m-mANSWERm-m50%m-
Formamcouple minmwhichmonemormbothmpartnersmaremaffectedmbymthemdisease,mthemchance mofmeachmchildmbeingm
heterozygousmaffectedmism50%.
Whichmof mthe mfollowingmstatementsmismtrue maboutmautosomalmrecessive mdisease?m-mANSWERm-
m Itm affects m bothm menm andm women m equally m -
Amcriterionmformrecognizingmtheminheritance mofmanmautosomalmrecessive mdiseasemismthatmmenmandmwome
nmwillmbe maffectedmbymitminmequalmmeasure.
Whatmismthe mproportionmof mthe mpopulationmaffectedmbymamdisease matmthe mspecificmpointminmtime?m-
m ANSWERm - m Prevalence m rate m -
Incidence mrate mrefersmtomhowmmanymnewmcasesmthere maremofmamdiseasemwithinmamperiodmofmtimemdivide
dmbymthemnumbermofmthe mpopulation'smindividuals.mThemprevalencemrate mismaffectedmbymthe mincidencemr
ate mand
howmlongmthemaffectedmpatientsmsurvive.mRiskmfactormismanythingmthatmcouldmleadmtomdisease.mRelative mris
kmismthe mmeasure mofmamriskmfactormeffect.
Whichmof mthemfollowingmshouldmbe mcompletedmwhenmamdiseasemismmultifactorialmbutmhasmamlargermgeneticmco
mponent?m-mANSWERm-mGetmamthoroughmfamilymhistorym-
Environmentmandmlifestyle mchoicesmmayminfluencemamdisease,mbutmdiseasesmsuchmasmbreastmcancermaremhe
reditary,msomthe mfamilymhistorymshouldmbe mreviewed.
Whichmactionmismthe mpurpose mof mthe minflammatorymprocess?m-mANSWERm-
mTom destroy mforeign, m invasive m microorganisms m -
The mgeneralmpurposemofminflammationmismtominitiate mthe mdestructionmofmforeignmorminvasivemorganisms.mAsmam
secondarymeffect,mitmincreasesmthe mhealingmandmimmune mresponse mprocessesmof mthe mbody.
Whatmcausesmedemamthatmoccursmduringmthe minflammatorymprocess?m-mANSWERm-
m Increased mvascularm permeability m -
Increasedmpressureminmthemvasculature msecondarymtomthemvasodilatorymeffectsmofminflammationmwillmresul
tminmthe mleakage mofmfluidmintomthirdmspacesm(i.e.,medema).
pg.m
4
m m m m m m m m
Questions with Correct Answers/ Updated
m m m m m
2024-2025.
Whichmof mthe mfollowingmismnotmamcellularmfunction?m-mANSWERm-mCombustionm-
Allmlivingmcellsmmustmabsorbmandmmetabolizemsourcesmofmenergy,mexcretemwastemandmby-
productsmofmthose mprocesses,mandmreproducemtomcontinue moffspring.mPyrotechnicmprocessesm(i.e.mcom
bustion) mare mgenerallymmetaphorical.
Whatmare mmitochondriamresponsible mfor?m-mANSWERm-mEnergymproductionm-
Mitochondriamare mthemenergymproductionmorganellesmof mthe mcellmresponsible mformthemmajormmetabolicmp
rocessesmofmcreatingmunitsmofmenergym(e.g.,mATP).
Whichmifmthe mfollowingmcanmcause medema?m-mANSWERm-mIncreasedmlymphaticmpressure m-
Decreasedmvascularmandmlymphaticmpressuremdoesmnotmgenerallymresultminmthemmovementmandmaccumula
tionmofmfluidmintomextravascularmspaces.mIncreasedmplasmampressureminmthe mvascularmresultsminmhyperten
sion,
while mincreasedmlymphaticmpressuremwillmreadilymresultminmthempoolingmof mlymphaticmdrainage mintomthirdm
spacesm(i.e.medema).
Whichmof mthe mfollowingmbestmdescribesmthe mresultmof mcellularmreproduction?m-mANSWERm-
m Twom diploid m cells, m calledm daughterm cells, m are m produced m -
The mresultantmoffspringmofmcellularmreproductionmismthemcreationmofmtwomdaughtermcellsmaftermthemcomple
tionmofmmitosismormmeiosis.mInterphasemismthe mlongestmperiodmthatmamcellmspendsmitsmlifemcycle mandmismth
e
preparatorymstage mbeforemcellularmreproduction.mAnaphasemismthemseparationmofmgeneticmmaterialsmtomthemop
posingmcentromeresmof mthe mcell,mfollowedmbymtelophase.
Whatmindicatesmhypokalemia?m-mANSWERm-mSerummKmdecreasesmtomlessmthanm3.5m-
pg.m
1
,Normalmserummpotassiumm(K),malsomknownmasmeukalemia,mismgenerallym3.5-
5.5mmEq/L,mwithmsome mvariationsmof m+/-m0.1-
0.3mmEq/Lmdependingmonmthe mlaboratorymtesting,mage,mandmothermpotentialmpatient-
specificmconditionsmandmvariations.mAlterationsminmfluidmvolumemormthemadministrationmofmamsupplementm
alone mdomnotmnecessarilymcontribute mtomthe mdevelopmentmof mhypokalemia.
Whatmismanmexample mof mbothmhyperplasiamandmhypertrophy?m-mANSWERm-
m Uterine m enlargementm due m tom pregnancy m -
Hyperplasiamismthe mincrease minmthe mnumbermofmcellsmof manmorganmormtissue,mwhile mhypertrophymismthe
enlargementmofmthe mcellsmofmamgivenmorganmormtissuemwithoutmamchangeminmthe mnumbermofmcells.mMuscle
m cellsm grow m andm enlargem (hypertrophy)minm responsemtomincreased mwork m orm stressm frommexercise m orm incr
eased
cardiovascularmpressures.mWhenmamlivermismdamagedmormpartmofmitmismremoved,mitmismthe mone minternalmsol
idmorganmcapable mofmregenerationmtomrepairmitselfmcreatingmmoremcellsm(hyperplasia).mDuringmpregnancy,m
the muterusmmustmbothmgrowmtomcreatemmore mcellsmandmenlarge mthosemexistingmcellsminmpreparationmformt
he msupportmof mamfetus.
Whatmregulatesmthe msodiummbalance?m- mANSWERm-mAldosterone m-
Aldosterone mismamregulatorymhormone mproducedmbymthemadrenalmglandsmtomaffectmthemkidneys'mregulationmof
m sodium, m potassium, m andm waterm excretion.
Whatmismthe malterationmifmthe mextracellularmfluidmvolume mismlessmthanmnormal?m-mANSWERm-mHypotonicm-
Inmhypotonicmstates,mthemcellmsizemismincreasedmasmamresultmofmwatermmovingmfrommthemextracellularmfluidm
(ECF) mintomthe mcellmtomincrease mintracellularmfluid.mIso-
m orm normotonicm states m are m am balanced m state m between m intra-
m andm extracellularm fluidmvolumes. mHypertonicm statesm cause mwatermtomshiftmoutmofm them cellm tom increase m t
he mECFmabove mnormal.
Whenminmexcess,mwhatmdombuffersmabsorb?m-mANSWERm-mHydrogenm-
Excessesmofmacidmormbase,mH+ mormhydroxylmionm(OH−),mare mabsorbedmformthempurpose mof mmaintainingmpHmho
meostasism(pHmof m7.38-7.42)
Whichmof mthe mfollowingmwouldmampatientmwithmmetabolicmacidosismhave?m-mANSWERm-
m pHm below m 7.35;m Bicarbonate m levelm below m 22m mEq/Lm -
The mdiagnosismofmmetabolicmacidosismrequiresmthatmthe mtestingmof marterialmbloodmfindsmthatmthe mpHmisml
essmthanm7.35mandmthe mbicarbonate mlevelmismlessmthanm22mmEq/L.
pg.m
2
,Whatmismthe mprocessmbymwhichmRNAmdirectsmthe msynthesismofmpolypeptides?m-mANSWERm-mTranslationm-
pg.m
3
, Translationmrefersmtomthismprocess.mTranscriptionmismanothermprocessminmwhichmRNAmismsynthesized.mMut
ationmmeansmgeneticmmaterialmhasmbeenmmodifiedmthroughminheritance.mCreationmismamtermmthatmdoesmn
otmapplyminmthismcontext.
Whatmismthe mrecurrence mrate mformautosomalmdominantmdisease?m-mANSWERm-m50%m-
Formamcouple minmwhichmonemormbothmpartnersmaremaffectedmbymthemdisease,mthemchance mofmeachmchildmbeingm
heterozygousmaffectedmism50%.
Whichmof mthe mfollowingmstatementsmismtrue maboutmautosomalmrecessive mdisease?m-mANSWERm-
m Itm affects m bothm menm andm women m equally m -
Amcriterionmformrecognizingmtheminheritance mofmanmautosomalmrecessive mdiseasemismthatmmenmandmwome
nmwillmbe maffectedmbymitminmequalmmeasure.
Whatmismthe mproportionmof mthe mpopulationmaffectedmbymamdisease matmthe mspecificmpointminmtime?m-
m ANSWERm - m Prevalence m rate m -
Incidence mrate mrefersmtomhowmmanymnewmcasesmthere maremofmamdiseasemwithinmamperiodmofmtimemdivide
dmbymthemnumbermofmthe mpopulation'smindividuals.mThemprevalencemrate mismaffectedmbymthe mincidencemr
ate mand
howmlongmthemaffectedmpatientsmsurvive.mRiskmfactormismanythingmthatmcouldmleadmtomdisease.mRelative mris
kmismthe mmeasure mofmamriskmfactormeffect.
Whichmof mthemfollowingmshouldmbe mcompletedmwhenmamdiseasemismmultifactorialmbutmhasmamlargermgeneticmco
mponent?m-mANSWERm-mGetmamthoroughmfamilymhistorym-
Environmentmandmlifestyle mchoicesmmayminfluencemamdisease,mbutmdiseasesmsuchmasmbreastmcancermaremhe
reditary,msomthe mfamilymhistorymshouldmbe mreviewed.
Whichmactionmismthe mpurpose mof mthe minflammatorymprocess?m-mANSWERm-
mTom destroy mforeign, m invasive m microorganisms m -
The mgeneralmpurposemofminflammationmismtominitiate mthe mdestructionmofmforeignmorminvasivemorganisms.mAsmam
secondarymeffect,mitmincreasesmthe mhealingmandmimmune mresponse mprocessesmof mthe mbody.
Whatmcausesmedemamthatmoccursmduringmthe minflammatorymprocess?m-mANSWERm-
m Increased mvascularm permeability m -
Increasedmpressureminmthemvasculature msecondarymtomthemvasodilatorymeffectsmofminflammationmwillmresul
tminmthe mleakage mofmfluidmintomthirdmspacesm(i.e.,medema).
pg.m
4
