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Test Bank For Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition by Strayer ISBN:9781451183900, All 34 Chapters Covered, Verified Latest Edition

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Test Bank For Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition by Strayer ISBN:9781451183900, All 34 Chapters Covered, Verified Latest Edition

Institution
Rubin\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\
Course
Rubin\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\

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TEST BANK FOR RUBIN’S PATHOLOGY:
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CLINICOPATHOLOGIC FOUNDATIONS OF
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MEDICINE 7TH EDITION ( WITH ANSWER
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KEY)
m




LATEST UPDATED EXAMINATION STUDY m m m m




GUIDE 2023
m m




GUARANTEED GRADE A+ m m




Chapter 1: Cell Adaptation, Injury and Death
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Ischemia and other toxic injuries increase the accumulation of intracellular calcium as a resu
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1. of:
A) release of stored calcium from the mitochondria.
m m m m m m




B) improved intracellular volume regulation. m m m




C) decreased influx across the cell membrane. m m m m m



D) attraction of calcium to fatty infiltrates. m m m m m



The patient is found to have liver disease, resulting in the removal of a lobe of his liver.
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2. Adaptation to the reduced size of the liver leads to _
m m of the remaining liver cells.
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A) metaplasia
B) organ atrophy m



C) compensatory hyperplasia m



D) physiologic hypertrophy m




A person eating peanuts starts choking and collapses. His airway obstruction is partially
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mcleared, but he remains hypoxic until he reaches the hospital. The prolonged cell hypoxia
m m m m m m m m m m m m m


3. caused a cerebral infarction and resulting
m m _ in the brain. m m m m m m



A) caspase activation m



B) coagulation necrosis m



C) rapid phagocytosis m



D) protein p53 deficiency m m



Bacteria and viruses cause cell damage by
m m m m m m , which is unique from the intracellular
m m m m m m


4. damage caused by other injurious agents.
m m m m m



A) disrupting the sodium/potassium ATPase pump m m m m



B) interrupting oxidative metabolism processes m m m



C) replicating and producing continued injury m m m m

,TEST BANK FOR RUBIN’S PATHOLOGY:
m m m m m




CLINICOPATHOLOGIC FOUNDATIONS OF
m m m m




MEDICINE 7TH EDITION ( WITH ANSWER
m m m m m m m




KEY)
m




LATEST UPDATED EXAMINATION STUDY
m m m m




GUIDE 2023
m m




GUARANTEED GRADE A+ m m




D) decreasing protein synthesis and function
m m m m



The patient has a prolonged interruption in arterial blood flow to his left kidney, causing
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5. hypoxic cell injury and the release of free radicals. Free radicals damage cells by:
m m m m m m m m m m m m m




A) destroying phospholipids in the cell membrane.
m m m m m



B) altering the immune response of the cell.
m m m m m m



C) disrupting calcium storage in the cell.
m m m m m



D) inactivation of enzymes and mitochondria.
m m m m

,TEST BANK FOR RUBIN’S PATHOLOGY:
m m m m m




CLINICOPATHOLOGIC FOUNDATIONS OF
m m m m




MEDICINE 7TH EDITION ( WITH ANSWER
m m m m m m m




KEY)
m




LATEST UPDATED EXAMINATION STUDY m m m m




GUIDE 2023
m m




GUARANTEED GRADE A+ m m




6. Injured cells have impaired flow of substances through the cell membrane as a result of:
m m m m m m m m m m m m m m




A) increased fat load. m m



B) altered permeability. m



C) altered glucose utilization.
m m



D) increased surface receptors. m m



7. Reversible adaptive intracellular responses are initiated by:
m m m m m m




A) stimulus overload. m



B) genetic mutations. m



C) chemical messengers. m



D) mitochondrial DNA. m



8. Injured cells become very swollen as a result of:
m m m m m m m m




A) increased cell protein synthesis. m m m



B) altered cell volume regulation.
m m m



C) passive entry of potassium into the cell.
m m m m m m



D) bleb formation in the plasma membrane.
m m m m m



A diabetic patient has impaired sensation, circulation, and oxygenation of his feet. He steps
m m m m m m m m m m m m m


mon a piece of glass, the wound does not heal, and the area tissue becomes necrotic. The
m m m m m m m m m m m m m m m m


mnecrotic
9. cell death is characterized by:
m m m m



A) rapid apoptosis.m



B) cellular rupture. m




C) shrinkage and collapse. m m



D) chronic inflammation. m



A 99-year-old woman has experienced the decline of cell function associated with age. A
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10. group of theories of cellular aging focus on programmed:
m m m m m m m m



A) changes with genetic influences.
m m m



B) elimination of cell receptor sites. m m m m



C) insufficient telomerase enzyme. m m

, TEST BANK FOR RUBIN’S PATHOLOGY:
m m m m m




CLINICOPATHOLOGIC FOUNDATIONS OF
m m m m




MEDICINE 7TH EDITION ( WITH ANSWER
m m m m m m m




KEY)
m




LATEST UPDATED EXAMINATION STUDY
m m m m




GUIDE 2023
m m




GUARANTEED GRADE A+ m m




D) DNA mutation or faulty repair.
m m m m




An 89-year-old female patient has experienced significant decreases in her mobility and
m m m m m m m m m m m


mstamina during a 3-week hospital stay for the treatment of a femoral head fracture. Which
m m m m m m m m m m m m m m


mof the following phenomena most likely accounts for the patients decrease in muscle
m m m m m m m m m m m m


mfunction
11. that underlies her reduced mobility?
m m m m




A) Impaired muscle cell metabolism resulting from metaplasia
m m m m m m



B) Dysplasia as a consequence of inflammation during bone remodeling
m m m m m m m m



C) Disuse atrophy of muscle cells during a prolonged period of immobility
m m m m m m m m m m

Written for

Institution
Rubin\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\
Course
Rubin\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\\

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Uploaded on
March 26, 2025
Number of pages
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Written in
2024/2025
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