TEST BANK FOR RUBIN'S PATHOLOGY- CLINICOPATHOLOGIC
FOUNDATIONS OF MEDICINE 7TH EDITION Author:DAVID S.
STRAYER, EMANUEL RUBIN All Chapters[1-34] Newest
Version Available in PDF
1|Page
,Chapter 1: Cell Adaptation, Injury And Death I
1. Of:
A) Release Of Stored Calcium From The Mitochondria.
B) Improved Intracellular Volume Regulation.
C) Decreased Influx Across The Cell Membrane.
D) Attraction Of Calcium To Fatty Infiltrates.
The Patient Is Found To Have Liver Disease, Resulting In The Removal Of A Lobe Of
His Liver.
2. Adaptation To The Reduced Size Of The Liver Leads To _ Of The Remaining Liver
Cells.
A) Metaplasia
B) Organ Atrophy
C) Compensatory Hyperplasia
D) Physiologic Hypertrophy
A Person Eating Peanuts Starts Choking And Collapses. His Airway Obstruction Is
Partially Cleared, But He Remains Hypoxic Until He Reaches The Hospital. The
Prolonged Cell Hypoxia
3. Caused A Cerebral Infarction And Resulting _ In The Brain.
A) Caspase Activation
B) Coagulation Necrosis
C) Rapid Phagocytosis
D) Protein P53 Deficiency
Bacteria And Viruses Cause Cell Damage By , Which Is Unique From The
Intracellular
4. Damage Caused By Other Injurious Agents.
A) Disrupting The Sodium/Potassium Atpase Pump
B) Interrupting Oxidative Metabolism Processes
C) Replicating And Producing Continued Injury
2|Page
, D) Decreasing Protein Synthesis And Function
The Patient Has A Prolonged Interruption In Arterial Blood Flow To His Left Kidney, Causing
5. Hypoxic Cell Injury And The Release Of Free Radicals. Free Radicals Damage Cells By:
A) Destroying Phospholipids In The Cell Membrane.
B) Altering The Immune Response Of The Cell.
C) Disrupting Calcium Storage In The Cell.
D) Inactivation Of Enzymes And Mitochondria.
3|Page
, TEST BANK FOR RUBIN’S PATHOLOGY:
CLINICOPATHOLOGIC FOUNDATIONS OF
MEDICINE 7TH EDITION ( WITH ANSWER
KEY)
LATEST UPDATED EXAMINATION STUDY
GUIDE 2023
GUARANTEED GRADE A+
6. Injured Cells Have Impaired Flow Of Substances Through The Cell Membrane As A Result O
A) Increased Fat Load.
B) Altered Permeability.
C) Altered Glucose Utilization.
D) Increased Surface Receptors.
7. Reversible Adaptive Intracellular Responses Are Initiated By:
A) Stimulus Overload.
B) Genetic Mutations.
C) Chemical Messengers.
D) Mitochondrial DNA.
8. Injured Cells Become Very Swollen As A Result Of:
A) Increased Cell Protein Synthesis.
B) Altered Cell Volume Regulation.
C) Passive Entry Of Potassium Into The Cell.
D) Bleb Formation In The Plasma Membrane.
A Diabetic Patient Has Impaired Sensation, Circulation, And Oxygenation Of His Feet. He
Steps On A Piece Of Glass, The Wound Does Not Heal, And The Area Tissue Becomes
Necrotic. The Necrotic
9. Cell Death Is Characterized By:
A) Rapid Apoptosis.
B) Cellular Rupture.
C) Shrinkage And Collapse.
D) Chronic Inflammation.
A 99-Year-Old Woman Has Experienced The Decline Of Cell Function Associated With Age
A
10. Group Of Theories Of Cellular Aging Focus On Programmed:
A) Changes With Genetic Influences.
B) Elimination Of Cell Receptor Sites.
4|Page
FOUNDATIONS OF MEDICINE 7TH EDITION Author:DAVID S.
STRAYER, EMANUEL RUBIN All Chapters[1-34] Newest
Version Available in PDF
1|Page
,Chapter 1: Cell Adaptation, Injury And Death I
1. Of:
A) Release Of Stored Calcium From The Mitochondria.
B) Improved Intracellular Volume Regulation.
C) Decreased Influx Across The Cell Membrane.
D) Attraction Of Calcium To Fatty Infiltrates.
The Patient Is Found To Have Liver Disease, Resulting In The Removal Of A Lobe Of
His Liver.
2. Adaptation To The Reduced Size Of The Liver Leads To _ Of The Remaining Liver
Cells.
A) Metaplasia
B) Organ Atrophy
C) Compensatory Hyperplasia
D) Physiologic Hypertrophy
A Person Eating Peanuts Starts Choking And Collapses. His Airway Obstruction Is
Partially Cleared, But He Remains Hypoxic Until He Reaches The Hospital. The
Prolonged Cell Hypoxia
3. Caused A Cerebral Infarction And Resulting _ In The Brain.
A) Caspase Activation
B) Coagulation Necrosis
C) Rapid Phagocytosis
D) Protein P53 Deficiency
Bacteria And Viruses Cause Cell Damage By , Which Is Unique From The
Intracellular
4. Damage Caused By Other Injurious Agents.
A) Disrupting The Sodium/Potassium Atpase Pump
B) Interrupting Oxidative Metabolism Processes
C) Replicating And Producing Continued Injury
2|Page
, D) Decreasing Protein Synthesis And Function
The Patient Has A Prolonged Interruption In Arterial Blood Flow To His Left Kidney, Causing
5. Hypoxic Cell Injury And The Release Of Free Radicals. Free Radicals Damage Cells By:
A) Destroying Phospholipids In The Cell Membrane.
B) Altering The Immune Response Of The Cell.
C) Disrupting Calcium Storage In The Cell.
D) Inactivation Of Enzymes And Mitochondria.
3|Page
, TEST BANK FOR RUBIN’S PATHOLOGY:
CLINICOPATHOLOGIC FOUNDATIONS OF
MEDICINE 7TH EDITION ( WITH ANSWER
KEY)
LATEST UPDATED EXAMINATION STUDY
GUIDE 2023
GUARANTEED GRADE A+
6. Injured Cells Have Impaired Flow Of Substances Through The Cell Membrane As A Result O
A) Increased Fat Load.
B) Altered Permeability.
C) Altered Glucose Utilization.
D) Increased Surface Receptors.
7. Reversible Adaptive Intracellular Responses Are Initiated By:
A) Stimulus Overload.
B) Genetic Mutations.
C) Chemical Messengers.
D) Mitochondrial DNA.
8. Injured Cells Become Very Swollen As A Result Of:
A) Increased Cell Protein Synthesis.
B) Altered Cell Volume Regulation.
C) Passive Entry Of Potassium Into The Cell.
D) Bleb Formation In The Plasma Membrane.
A Diabetic Patient Has Impaired Sensation, Circulation, And Oxygenation Of His Feet. He
Steps On A Piece Of Glass, The Wound Does Not Heal, And The Area Tissue Becomes
Necrotic. The Necrotic
9. Cell Death Is Characterized By:
A) Rapid Apoptosis.
B) Cellular Rupture.
C) Shrinkage And Collapse.
D) Chronic Inflammation.
A 99-Year-Old Woman Has Experienced The Decline Of Cell Function Associated With Age
A
10. Group Of Theories Of Cellular Aging Focus On Programmed:
A) Changes With Genetic Influences.
B) Elimination Of Cell Receptor Sites.
4|Page
