Test Bank For Rubin's Pathology: Clinicopathologic Foundations of Medicine Seventh Edition by David S. Strayer ||ISBN 978-1451183900|| Chapter 1-34||Complete Guide A+

TEST BANK FOR RUBIN’S PATHOLOGY: CL
c c c c c




INICOPATHOLOGIC FOUNDATIONS OFME c c c




DICINE 7TH EDITION ( WITH ANSWERKEY)
c c c c c c




LATEST UPDATED EXAMINATION STUDY
c c c c




GUIDE 2023 c




GUARANTEED GRADEA+ c c




Chapterc1:cCellcAdaptation,cInjurycandcDeath

Ischemiacandcotherctoxiccinjuriescincreasecthecaccumulationcofcintracellularccalciumcascacresult
c

1. of:
A) releasecofcstoredccalciumcfromcthecmitochondria.
B) improvedcintracellularcvolumecregulation.
C) decreasedcinfluxcacrossctheccellcmembrane.
D) attractioncofccalciumctocfattycinfiltrates.
Thecpatientciscfoundctochaveclivercdisease,cresultingcincthecremovalcofcaclobecofchiscliver.
2. Adaptationctocthecreducedcsizecofctheclivercleadsctoc_ ofcthecremainingcliverccells.
A) metaplasia
B) organcatrophy
C) compensatorychyperplasia
D) physiologicchypertrophy
Acpersonceatingcpeanutscstartscchokingcandccollapses.cHiscairwaycobstructionciscpartiallyccl
eared,cbutchecremainschypoxiccuntilchecreachescthechospital.cThecprolongedccellchypoxia
3. causedcaccerebralcinfarctioncandcresulting _cincthecbrain.
A) caspasecactivation
B) coagulationcnecrosis
C) rapidcphagocytosis
D) proteincp53cdeficiency
Bacteriacandcvirusesccauseccellcdamagecby ,cwhichciscuniquecfromcthecintracellular
4. damageccausedcbycothercinjuriouscagents.
A) disruptingcthecsodium/potassiumcATPasecpump
B) interruptingcoxidativecmetabolismcprocesses
C) replicatingcandcproducingccontinuedcinjury

,TEST BANK FOR RUBIN’S PATHOLOGY: CL
c c c c c




INICOPATHOLOGIC FOUNDATIONS OFME c c c




DICINE 7TH EDITION ( WITH ANSWERKEY)
c c c c c c




LATEST UPDATED EXAMINATION STUDY
c c c c




GUIDE 2023 c




GUARANTEED GRADEA+ c c




D) decreasingcproteincsynthesiscandcfunction
Thecpatientchascacprolongedcinterruptioncincarterialcbloodcflowctochiscleftckidney,ccausing
5. hypoxicccellcinjurycandcthecreleasecofcfreecradicals.cFreecradicalscdamageccellscby:
A) destroyingcphospholipidscinctheccellcmembrane.
B) alteringcthecimmunecresponsecofctheccell.
C) disruptingccalciumcstoragecinctheccell.
D) inactivationcofcenzymescandcmitochondria.

,TEST BANK FOR RUBIN’S PATHOLOGY: CL
c c c c c




INICOPATHOLOGIC FOUNDATIONS OFME c c c




DICINE 7TH EDITION ( WITH ANSWERKEY)
c c c c c c




LATEST UPDATED EXAMINATION STUDY
c c c c




GUIDE 2023 c




GUARANTEED GRADEA+ c c




6. Injuredccellschavecimpairedcflowcofcsubstancescthroughctheccellcmembranecascacresultcof:
A) increasedcfatcload.
B) alteredcpermeability.
C) alteredcglucosecutilization.
D) increasedcsurfacecreceptors.
7. Reversiblecadaptivecintracellularcresponsescarecinitiatedcby:
A) stimuluscoverload.
B) geneticcmutations.
C) chemicalcmessengers.
D) mitochondrialcDNA.
8. Injuredccellscbecomecverycswollencascacresultcof:
A) increasedccellcproteincsynthesis.
B) alteredccellcvolumecregulation.
C) passivecentrycofcpotassiumcintoctheccell.
D) blebcformationcincthecplasmacmembrane.
Acdiabeticcpatientchascimpairedcsensation,ccirculation,candcoxygenationcofchiscfeet.cHecstepsconcac
piececofcglass,cthecwoundcdoescnotcheal,candcthecareactissuecbecomescnecrotic.cThecnecrotic
9. cellcdeathcisccharacterizedcby:
A) rapidcapoptosis.
B) cellularcrupture.
C) shrinkagecandccollapse.
D) chroniccinflammation.
Ac99-year-oldcwomanchascexperiencedcthecdeclinecofccellcfunctioncassociatedcwithcage.cA
10. groupcofctheoriescofccellularcagingcfocusconcprogrammed:
A) changescwithcgeneticcinfluences.
B) eliminationcofccellcreceptorcsites.
C) insufficientctelomerasecenzyme.

, TEST BANK FOR RUBIN’S PATHOLOGY: CL
c c c c c




INICOPATHOLOGIC FOUNDATIONS OFME c c c




DICINE 7TH EDITION ( WITH ANSWERKEY)
c c c c c c




LATEST UPDATED EXAMINATION STUDY
c c c c




GUIDE 2023 c




GUARANTEED GRADEA+ c c




D) DNAcmutationcorcfaultycrepair.
Anc89-year-
oldcfemalecpatientchascexperiencedcsignificantcdecreasescinchercmobilitycandcstaminacduringcac
3-
weekchospitalcstaycforcthectreatmentcofcacfemoralcheadcfracture.cWhichcofcthecfollowingcpheno
menacmostclikelycaccountscforcthecpatientscdecreasecincmusclecfunction
11. thatcunderlieschercreducedcmobility?
A) Impairedcmuscleccellcmetabolismcresultingcfromcmetaplasia
B) Dysplasiacascacconsequencecofcinflammationcduringcbonecremodeling
C) Disusecatrophycofcmuscleccellscduringcacprolongedcperiodcofcimmobility