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Test Bank For Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives Third Edition by Theresa Capriotti, Chapters 1-46, Complete Guide A+

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Test Bank For Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives Third Edition by Theresa Capriotti, Chapters 1-46, Complete Guide A+ Test Bank For Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives Third Edition by Theresa Capriotti, Chapters 1-46, Complete Guide A+ Test Bank For Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives Third Edition by Theresa Capriotti, Chapters 1-46, Complete Guide A+ Test Bank For Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives Third Edition by Theresa Capriotti, Chapters 1-46, Complete Guide A+ Test Bank For Davis Advantage for Pathophysiology: Introductory Concepts and Clinical Perspectives Third Edition by Theresa Capriotti, Chapters 1-46, Complete Guide A+

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Test Bank For Davis Advantage for Pathophysiology:
IntroductoryConcepts and Clinical Perspectives 3e
Capriotti ; Complete Solution.


Chapter \1, \The \Cell \in \Health \and \Illness Complete \Chapters \
Multiple \Choice
Identify \the \choice \that \best \completes \the \statement \or \answers \the Answers \Included
\question.


1. \ \ Which \statement \regarding \the \sodium-potassium \pump \is \correct?
1. The \cell’s \plasma \membrane \is \more \soluble \to \sodium \ions \than \potassium
\ions.
2. The \concentration \of \sodium \ions \should \be \higher \inside \the \cell \compartment.
3. The \concentration \of \potassium \ions \should \be \higher \outside \the \cell
\compartment.
4. Active \transport \involves \pumping \out \three \sodium \ions \and \pumping \in \two
\potassium \ions.




2. \ Which \cellular \function \generates \the \same \amount \of \energy \in \the \absence \of \oxygen \as \it
\does \in \the \presence \of \oxygen?
1. Dissipation \of \pyruvic \acid
2. Initiation \of \the \citric \acid \cycle
3. Activation \of \acetyl-coenzyme \A
4. Creation \of \acidosis \via \lactic \acid


3. \ \ How \many \adenosine \triphosphates \(ATPs) \are \produced \in \aerobic \energy \metabolism?
1. 2
2. 3
3. 34
4. 53


4. \ \ Which \cell \organelles \differ \in \their \number \according \to \the \cell’s \energy \needs?
1. Ribosomes
2. Mitochondria
3. Ribonucleic \acids
4. Deoxyribonucleic \acids

,5. \ \ Which \option \best \explains \why \more \energy \is \produced \when \a \person \is \exercising?
1. Exercise \causes \an \increase \in \the \synthesis \of \protein.
2. There \is \an \increase \in \the \production \of \pyruvic \acid \in \the \cells.
3. The \conversion \of \pyruvic \acid \to \lactic \acid \is \increased \by \exercise.
4. Muscle \cells \have \more \mitochondria \to \meet \energy \demands.
024

, 6. \ \ When \does \ribosomal \protein \synthesis \cease?
1. During \endoplasmic \reticulum \stress
2. During \the \synthesis \of \adenosine \triphosphate \(ATP)
3. During \severe \hypoxia
4. During \the \processing \of \prohormone


7. \ \ Which \cellular \components \are \responsible \for \propelling \mucus \and \inhaled \debris \out \of \the
\lungs?
1. Cilia
2. Microfilaments
3. Secretory \vesicles
4. Endoplasmic \reticula


8. \ \ What \are \the \key \proteins \in \the \contractile \units \of \the \muscle \cells?
1. Actin \and \myosin
2. Prohormone \and \tubulin
3. Tubulin \and \actin
4. Myosin \and \prohormone


9. \ \ Which \deficiency \causes \Tay-Sachs \disease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomal \enzymes


\ \ 10. \ \ Which \of \the \following \is \a \characteristic \of \adrenoleukodystrophy?
1. Accumulation \of \ganglioside
2. Cessation \of \ribosomal \protein \synthesis
3. Acceleration \of \cellular \proteasome \activity
4. Accumulation \of \long-chain \fatty \acids \in \the \nervous \system


\ \ 11. \ Which \statement \regarding \endoplasmic \reticulum \(ER) \stress \is \correct?
1. During \ER \stress, \proteins \are \rapidly \degraded.
2. During \ER \stress, \lipids \cannot \travel \to \their \proper \intracellular \locations.
3. During \ER \stress, \long-chain \fatty \acids \accumulate \in \the \nervous \system.
4. During \ER \stress, \nondegraded \substances \accumulate \in \the \cells.

, \ \ 12. \ Which \statement \regarding \mitochondria \is \correct?
1. Mitochondrial \DNA \is \identical \to \nuclear \DNA.
2. Mitochondria \reproduce \in \response \to \an \increased \need \for \ATP.
3. Mitochondria \are \primarily \obtained \from \sperm \during \human \fertilization.
4. Mitochondrial \DNA \is \immune \to \the \effects \of \free \radicals.


\ 13. \ A \newborn \patient \exhibits \characteristics \of \severe \physical \deformities. \Which \cellular
\component \is \examined \to \determine \the \cause \and \probability \of \the \disease \being \genetically
\transferred?
1. Transfer \RNA
2. Ribosomal \RNA
3. Double \helix \of \DNA
4. Mitochondrial \DNA


\ 14. \ A \hiker \experiences \muscle \pain \and \acidosis \while \ascending \a \high, \steep \mountain. \Which
\option \is \the \reason \for \these \manifestations?
1. Cellular \hypoxia
2. Autolysis
3. Heterolysis
4. Cellular \edema


\ \ 15. \ \ Which \factor \accounts \for \DNA’s \unique \molecular \ability \to \replicate?
1. The \pairing \of \nitrogenous \bases
2. The \presence \of \pyrimidine \bases
3. The \presence \of \nucleotides
4. The \nitrogenous \base \and \phosphate \bond


\ \ 16. \ How \many \nitrogenous \bases \compose \a \single \codon?
1. 2
2. 3
3. 4
4. 5


\ \ 17. \ Which \components \form \the \structure \of \DNA?
1. Nucleotides
2. Amino \acids

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