TEST BANK FOR DAVIS ADVANTAGE
k k k k k
FOR PATHOPHYSIOLOGY 2ND EDITI
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written by
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Chapter k1,kThekCellkinkHealthkandkIllness
MultiplekChoice
Identifykthekchoicekthatkbestkcompleteskthekstatementkorkanswerskthekquestion.
1. Whichk statement k regardingk thek sodium–potassiumk pumpk isk correct?
1. Thek cell’sk plasmak membranek isk morek solublek tok sodiumk ionsk thank potassiumk ions.
2. Thek concentrationk of k sodiumk ionsk should k bek higherk insidek thek cellk compartment.
3. Thek concentrationk of k potassiumk ionsk should kbekhigherk outsidek thek cellk
compartment.
4. Thek activek transport k involvesk pumpingk out k threek sodiumk ionsk and k pumpingk ink two
potassiumk ions.
2. Ink thek absencek ofk oxygen,kwhichk cellulark functionk createsktheksamek amount kof kenergyk ask isk created k ink t
hek presencek of k oxygen?
1. Dissipationk of k pyruvick acid
2. Initiationk of k thek citrick acid k cycle
3. Activationkof k acetyl-coenzymekA
4. Creationk of k acidosisk viak lactick acid
3. How kmanyk adenosinek triphosphatesk (ATPs)k arek produced k ink aerobickenergyk metabolism?
1.k k 2
2.k k 3
3.k k 34
4.k k 53
4. Whichk cellk organellesk differk ink theirk numberk accordingk tok thek cell’sk energyk needs?
1. Ribosomes
2. Mitochondria
3. Ribonucleick acids
4. Deoxyribonucleickacids
5. Whichk optionk best k supportsk thek reasonk morek energyk isk produced k whenk ak personk isk exercising?
1. Exercisek causesk ank increasek ink thek synthesisk of k protein.
2. Therek isk ank increasek ink thek productionk of k pyruvick acid k ink thek cells.
3. Thek conversionk of k pyruvick acid k tok lactick acid k isk increased k byk exercise.
4. Musclek cellsk havek morek mitochondriak tok meet k energyk demands.
6. Whenk doesk ribosomalk proteink synthesisk cease?
1. Duringk endoplasmick reticulumk stress
2. Duringk thek synthesisk of k adenosinek triphosphatek (ATP)
3. Duringk ak severek hypoxick state
4. Duringk thek processingk of k prohormone
7. Whichk cellulark organellesk arek responsiblek fork propellingk mucusk and k inhaled k debrisk out k of k thek lungs?
1. Cilia
2. Microfilaments
Copyright k©k2020kF.kA.kDavis kCompany
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3. Secretoryk vesicles
4. Endoplasmick reticula
8.k k Whichk arek thek keyk proteinsk ink thek contractilek unitsk of k thek musclek cells?
1. Actink and k myosin
2. Prohormonek and k tubulin
3. Tubulink and k actin
4. Myosink and k prohormone
9.k k Whichk deficiencyk causeskTay–Sachsk disease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomalk enzymes
k 10.k Whichk isk ak characteristick of k adrenoleukodystrophy?
1. Accumulationk of k ganglioside
2. Cessationk of k ribosomalk proteink synthesis
3. Accelerationk of k cellulark proteasomek activity
4. Accumulationk of k long-chaink fattyk acidsk ink thek nervousk system
k 11.k Whichk statement k regardingk endoplasmick reticulumk (ER)k stressk isk correct?
1. Duringk ERk stress,k proteinsk arek rapidlyk degraded.
2. Duringk ERk stress,k lipidsk cannot k travelk tok theirk properk intracellulark locations.
3. Duringk ERk stress,k long-chaink fattyk acidsk accumulatek ink thek nervousk system.
4. Duringk ERk stress,k nondegraded k substancesk accumulatek ink thek cells.
k 12.k Akclient k isk diagnosed kwithktypek1k diabeteskmellitus.kAt kak cellulark level,k whichk functionkisk likelyk tok bekin
volved?
1. Inabilityk of k ribosomesk tok producek ak specifick typek of k protein
2. Incorrect k processingk of k ak proteink byk thek Golgik apparatus
3. Stagnationk of k ak previouslyk dynamick actionk ink microtubules
4. Obstructionk of k thek smoothk endoplasmick reticulum
k 13.k Aknewbornk patient k exhibitsk characteristicsk of k severek physicalk deformities.kWhichk cellulark component k i
sk examined k tok determinek thek causek and k probabilityk of k thek diseasek beingk geneticallyk transferred?
1. TransferkRNA
2. Ribosomalk RNA
3. Doublek helixk of k DNA
4. Mitochondrialk DNA
k 14.k Akhikerk experiencesk musclek paink and k acidosisk whilek ascendingkakmountaink duringk aklong,k steepkc
limb.k Whichk isk thek reasonk fork thesek manifestations?
1. Cellulark hypoxia
2. Autolysis
3. Heterolysis
4. Cellulark edema
Copyright k©k2020kF.kA.kDavis kCompany
Downloadedk by:kSophiiek|
Distribution k ofk this k documentk is k illegal
k k k k k
FOR PATHOPHYSIOLOGY 2ND EDITI
k k k
ON BY CAPRIO
k k
written by
k
Sophiie
,www.stuvia.com
Downloadedk by:kSophiiek|
Distribution k ofk this k documentk is k illegal
, Stuvia.comk -
k Thek Marketplacek to k Buy k and k Sell k your k Study k Material
Chapter k1,kThekCellkinkHealthkandkIllness
MultiplekChoice
Identifykthekchoicekthatkbestkcompleteskthekstatementkorkanswerskthekquestion.
1. Whichk statement k regardingk thek sodium–potassiumk pumpk isk correct?
1. Thek cell’sk plasmak membranek isk morek solublek tok sodiumk ionsk thank potassiumk ions.
2. Thek concentrationk of k sodiumk ionsk should k bek higherk insidek thek cellk compartment.
3. Thek concentrationk of k potassiumk ionsk should kbekhigherk outsidek thek cellk
compartment.
4. Thek activek transport k involvesk pumpingk out k threek sodiumk ionsk and k pumpingk ink two
potassiumk ions.
2. Ink thek absencek ofk oxygen,kwhichk cellulark functionk createsktheksamek amount kof kenergyk ask isk created k ink t
hek presencek of k oxygen?
1. Dissipationk of k pyruvick acid
2. Initiationk of k thek citrick acid k cycle
3. Activationkof k acetyl-coenzymekA
4. Creationk of k acidosisk viak lactick acid
3. How kmanyk adenosinek triphosphatesk (ATPs)k arek produced k ink aerobickenergyk metabolism?
1.k k 2
2.k k 3
3.k k 34
4.k k 53
4. Whichk cellk organellesk differk ink theirk numberk accordingk tok thek cell’sk energyk needs?
1. Ribosomes
2. Mitochondria
3. Ribonucleick acids
4. Deoxyribonucleickacids
5. Whichk optionk best k supportsk thek reasonk morek energyk isk produced k whenk ak personk isk exercising?
1. Exercisek causesk ank increasek ink thek synthesisk of k protein.
2. Therek isk ank increasek ink thek productionk of k pyruvick acid k ink thek cells.
3. Thek conversionk of k pyruvick acid k tok lactick acid k isk increased k byk exercise.
4. Musclek cellsk havek morek mitochondriak tok meet k energyk demands.
6. Whenk doesk ribosomalk proteink synthesisk cease?
1. Duringk endoplasmick reticulumk stress
2. Duringk thek synthesisk of k adenosinek triphosphatek (ATP)
3. Duringk ak severek hypoxick state
4. Duringk thek processingk of k prohormone
7. Whichk cellulark organellesk arek responsiblek fork propellingk mucusk and k inhaled k debrisk out k of k thek lungs?
1. Cilia
2. Microfilaments
Copyright k©k2020kF.kA.kDavis kCompany
Downloadedk by:kSophiiek|
Distribution k ofk this k documentk is k illegal
, Stuvia.comk -
k Thek Marketplacek to k Buy k and k Sell k your k Study k Material
3. Secretoryk vesicles
4. Endoplasmick reticula
8.k k Whichk arek thek keyk proteinsk ink thek contractilek unitsk of k thek musclek cells?
1. Actink and k myosin
2. Prohormonek and k tubulin
3. Tubulink and k actin
4. Myosink and k prohormone
9.k k Whichk deficiencyk causeskTay–Sachsk disease?
1. Proteasome
2. Peroxisome
3. Macrophage
4. Lysosomalk enzymes
k 10.k Whichk isk ak characteristick of k adrenoleukodystrophy?
1. Accumulationk of k ganglioside
2. Cessationk of k ribosomalk proteink synthesis
3. Accelerationk of k cellulark proteasomek activity
4. Accumulationk of k long-chaink fattyk acidsk ink thek nervousk system
k 11.k Whichk statement k regardingk endoplasmick reticulumk (ER)k stressk isk correct?
1. Duringk ERk stress,k proteinsk arek rapidlyk degraded.
2. Duringk ERk stress,k lipidsk cannot k travelk tok theirk properk intracellulark locations.
3. Duringk ERk stress,k long-chaink fattyk acidsk accumulatek ink thek nervousk system.
4. Duringk ERk stress,k nondegraded k substancesk accumulatek ink thek cells.
k 12.k Akclient k isk diagnosed kwithktypek1k diabeteskmellitus.kAt kak cellulark level,k whichk functionkisk likelyk tok bekin
volved?
1. Inabilityk of k ribosomesk tok producek ak specifick typek of k protein
2. Incorrect k processingk of k ak proteink byk thek Golgik apparatus
3. Stagnationk of k ak previouslyk dynamick actionk ink microtubules
4. Obstructionk of k thek smoothk endoplasmick reticulum
k 13.k Aknewbornk patient k exhibitsk characteristicsk of k severek physicalk deformities.kWhichk cellulark component k i
sk examined k tok determinek thek causek and k probabilityk of k thek diseasek beingk geneticallyk transferred?
1. TransferkRNA
2. Ribosomalk RNA
3. Doublek helixk of k DNA
4. Mitochondrialk DNA
k 14.k Akhikerk experiencesk musclek paink and k acidosisk whilek ascendingkakmountaink duringk aklong,k steepkc
limb.k Whichk isk thek reasonk fork thesek manifestations?
1. Cellulark hypoxia
2. Autolysis
3. Heterolysis
4. Cellulark edema
Copyright k©k2020kF.kA.kDavis kCompany
Downloadedk by:kSophiiek|
Distribution k ofk this k documentk is k illegal
