TO kGET kALL kCHAPTERS kEMAIL kME kAT>>>>> k k k
Test bank for MCQs in Pediatrics Review of Nelson Textbook of
k k k k k k k k k k
k Pediatrics 20 Edition
k k
, TO kGET kALL kCHAPTERS kEMAIL kME kAT>>>>> k k k
1. Which kof k the k following k statements k regarding k foster k care k is k true?
□Akpermanency kplankmust kbe kmade kforkakchildkin kfosterkcare knoklaterkthan k12kmokfrom kthe kchild's kentrykintokcare
□Akminoritykofkchildrenkinkfosterkcarekhavekakhistorykofkabusekorkneglect
□Thekmissionkofkfosterkcarekisktoksafelykcarekforkchildrenkwhilekprovidingkservicesktokfamiliesktokpromotekreunification
□Mostk(>70%)kofkchildrenkinkfosterkcarekarekreunitedkwithktheirkfamilies
■ A kand kC
description kThe kmission kof kfoster kcare kis kto kprovide kfor kthe khealth, ksafety, kand kwell-being kof kchildren kwhile kassisting
ktheir k families kwith kservices kto kpromote kreunification. kChildren kentering kfoster kcare khave kfrequently kexperienced kearly
kchildhood k trauma. kMore kthan k70% khave ka khistory kof kabuse, kneglect, kor kboth. kOnly kabout k50% kof kchildren kachieve
kreunification. kIn kthe k USA, kthe kAdoption kand kSafe kFamilies kAct k(P.L. k105-89) kpassed kin k1997 krequires kthat ka
kpermanency kplan kbe kmade kfor k eachkchildknoklaterkthank12 kmokafterkentry kto kfoster kcarekand kthatka kpetitionkto kterminate
kparental krightsktypically kmustkbe kfiled k when ka kchild khas kbeen kin kfoster kcare kfor kat kleast k15 kof kthe kprevious k22 kmo.
k(See kChapter k35, kpage k134, kand ke35-1.)
2. A k4 kyr kold kgirl kis kadmitted kto kthe khospital kfor kher kthird kevaluation kfor kvaginal kbleeding. kThe
mother knoted kbright kred kblood kon kthe kchild's kunderwear. kPrevious kexaminations krevealed ka k normal
k4 kyr kold kgirl, kTanner kstage k1, kwith knormal kexternal kgenitalia. kPelvic kultrasound kresults k were knormal,
kas kwas kthe kserum kestradiol klevel. kThe khemoglobin kand kplatelet kcounts kwere k normal, kas kwere kthe
kbleeding ktime kand kcoagulation kstudies. kFindings kon kpelvic kexamination k conducted kunder
kanesthesia kalso kwere knormal. kThe knext kstep kin kthe kexamination kis kto:
■ Determine kthe kblood ktype kof kthe kblood kon kthe kunderwear
□Interrogate k the k father
□Isolate kthe kparents kand kchild
□Determine kvon kWillebrandkfactor klevels
, □Measure kfibronectinkinkthekvagina
description kConsideration kof kfactitious kdisorder kby kproxy kshould kbe ktriggered kwhen kthe kreported ksymptoms kare
krepeatedly k notedkbykonlykonekparent,kappropriatektestingkfailsktokconfirmkakdiagnosis,kandkseeminglykappropriate
ktreatmentkiskineffective.
Atktimes,kthekchild'sksymptoms,ktheirkcourse,korkthekresponsektoktreatmentkmaykbekincompatiblekwithkanykrecognizedkdisease.
k Preverbalkchildrenkarekusuallykinvolved.kBleedingkiskakparticularlykcommonkpresentation.kThiskmaykbekcausedkbykaddingkdyes
k k to ksamples, kadding kblood k(e.g., kfrom kthe kmother) kto kthe kchild's ksample, kor kgiving kthe kchild kan kanticoagulant k(e.g.,
kwarfarin). k (See kChapter k37, kpage k146.)
3. Munchausen ksyndrome kby kproxy kis kcharacterized kby kall kof k the kfollowing kEXCEPT:
□Motherkwho kappears kdevoted kand kwins koverkmembers kofkcarekteam
□Multiple k hospitalizations k and k investigations k without k diagnosis
□Symptomskonkhistorykbutknotkwitnessedkbykmedicalkteam
■ Symptomskoccurringkinkpresencekofkdifferentkcaregiversk(e.g.,kwhilekmotherkiskoutkofktown)
□Use kof kmedications kor ktoxins
description kSymptoms kin kyoung kchildren kare kmostly kassociated kwith kproximity kof kthe koffending kcaregiver kto kthe kchild.
kThe k mother kmay kpresent kas ka kdevoted kor keven kmodel kparent kwho kforms kclose krelationships kwith kmembers kof kthe
khealth kcare k team. kWhile kappearing kvery kinterested kin kher kchild's kcondition, kshe kmay kbe krelatively kdistant
kemotionally. k(See kChapter k37, k kpage k146.)
4. Which kstatement k is kfalse?
■ Malnutrition kis kthe ksecond kleading kcause kof kacquired kimmune kdeficiency kworldwide kbehind kHIV kinfection
□Zinc kis kimportant kinkimmunekfunction kand klinear kgrowth
□Kwashiorkor kand kmarasmus kare krare kin kdeveloped kcountries
□The kWestern kdiet kis kassociated kwith kincreased knoncommunicable kdisease
descriptionkTheksignificantkglobalkburdenkofkmalnutritionkandkundernutritionkiskthekleadingkworldwidekcausekofkacquired
k immunodeficiencykandkthekmajorkunderlyingkfactorkforkmorbiditykandkmortalitykgloballykforkchildrenk<5kyrkofkage.kZinckis
ka k micronutrientkthatksupportskmultiplekmetabolickfunctionskinkthekbody,kiskessentialkforknormalkimmunekfunctioning,kandkis
k requiredktoksupportklinearkgrowth;kzinckdeficiencykiskassociatedkwithkimpairedkimmunekfunctioningkandkpoorklinearkgrowth.
kIn k parallelktokthekriskkforknutrientkandkenergykdeficiencies,kissueskrelatingktokexcesseskposekimportantkchallengeskbecause
kofktheir k negative khealth keffects, ksuch kas kobesity kor kcardiovascular kdisease krisk kfactors. kThe knutrition ktransition
kunder kway kin kthe
, developing kworld kfrom ktraditional kdiets kto kthe kWestern kdiet khas kbeen kassociated kwith kincreases kin knoncommunicable
k diseases, koften kcoexisting kwith kundernutrition kand kmalnutrition, kobserved ksometimes kin kthe ksame kcommunities kor keven kthe
k same kfamilies. k(See ke41-1.)
5. Components k of k energy k expenditure k in kchildren k include:
□Thermalkeffectkofkfood
□Basal kmetabolic k rate
□Energy kfor kphysical kactivity
□Energyktoksupportkgrowth
■ All kof kthe kabove
description kThe k3 kcomponents kof kenergy kexpenditure kin kadults kare kthe kbasal kmetabolic krate, kthe kthermal keffect
kof kfood k (energy krequired kfor kdigestion kand kabsorption), kand kenergy kfor kphysical kactivity. kAdditional kenergy kintake
kand kexpenditure k are krequired kto ksupport kgrowth kand kdevelopment kfor kchildren. k(See ke41-4.)
6. Which kof kthe kfollowing kclinical kscenarios kincreases kthe krisk kof kvitamin kA kdeficiency?
□Vegetarian k diet
□Chronic k intestinal k disorders
□Zinc kdeficiency
■ B kand kC
□Allkofkthekabove
description kVitamin kA kis kan kessential kmicronutrient kbecause kit kcannot kbe kbiogenerated kde knovo kby kanimals. kIt
kmust kbe k obtained kfrom kplants kin kthe kform kof kprovitamin-A kcarotenoids. kIn kthe kUSA, kgrains kand kvegetables ksupply
kapproximately k 55% kand kdairy kand kmeat kproducts ksupply kapproximately k30% kof kvitamin kA kintake kfrom kfood.
kVitamin kA kand kthe k provitamins-Akarekfatksoluble,kandktheirkabsorptionkdependskonkthekpresencekofkadequateklipidkand
kproteinkwithinkthekmeal.
Chronic kintestinal kdisorders kor klipid kmalabsorption ksyndromes kcan kresult kin kvitamin kA kdeficiency. kIn kdeveloping kcountries,
k subclinicalkorkclinicalkzinckdeficiencykcankincreasekthekriskkofkvitaminkAkdeficiency.kTherekiskalsoksomekevidencekofkmarginal
k zinc kintakes k in kchildren kin kthe kUSA. k(See kChapter k45, k page k188.)
7. Which kstatement k about k vitamin k A k toxicity k is k NOT k true?
Test bank for MCQs in Pediatrics Review of Nelson Textbook of
k k k k k k k k k k
k Pediatrics 20 Edition
k k
, TO kGET kALL kCHAPTERS kEMAIL kME kAT>>>>> k k k
1. Which kof k the k following k statements k regarding k foster k care k is k true?
□Akpermanency kplankmust kbe kmade kforkakchildkin kfosterkcare knoklaterkthan k12kmokfrom kthe kchild's kentrykintokcare
□Akminoritykofkchildrenkinkfosterkcarekhavekakhistorykofkabusekorkneglect
□Thekmissionkofkfosterkcarekisktoksafelykcarekforkchildrenkwhilekprovidingkservicesktokfamiliesktokpromotekreunification
□Mostk(>70%)kofkchildrenkinkfosterkcarekarekreunitedkwithktheirkfamilies
■ A kand kC
description kThe kmission kof kfoster kcare kis kto kprovide kfor kthe khealth, ksafety, kand kwell-being kof kchildren kwhile kassisting
ktheir k families kwith kservices kto kpromote kreunification. kChildren kentering kfoster kcare khave kfrequently kexperienced kearly
kchildhood k trauma. kMore kthan k70% khave ka khistory kof kabuse, kneglect, kor kboth. kOnly kabout k50% kof kchildren kachieve
kreunification. kIn kthe k USA, kthe kAdoption kand kSafe kFamilies kAct k(P.L. k105-89) kpassed kin k1997 krequires kthat ka
kpermanency kplan kbe kmade kfor k eachkchildknoklaterkthank12 kmokafterkentry kto kfoster kcarekand kthatka kpetitionkto kterminate
kparental krightsktypically kmustkbe kfiled k when ka kchild khas kbeen kin kfoster kcare kfor kat kleast k15 kof kthe kprevious k22 kmo.
k(See kChapter k35, kpage k134, kand ke35-1.)
2. A k4 kyr kold kgirl kis kadmitted kto kthe khospital kfor kher kthird kevaluation kfor kvaginal kbleeding. kThe
mother knoted kbright kred kblood kon kthe kchild's kunderwear. kPrevious kexaminations krevealed ka k normal
k4 kyr kold kgirl, kTanner kstage k1, kwith knormal kexternal kgenitalia. kPelvic kultrasound kresults k were knormal,
kas kwas kthe kserum kestradiol klevel. kThe khemoglobin kand kplatelet kcounts kwere k normal, kas kwere kthe
kbleeding ktime kand kcoagulation kstudies. kFindings kon kpelvic kexamination k conducted kunder
kanesthesia kalso kwere knormal. kThe knext kstep kin kthe kexamination kis kto:
■ Determine kthe kblood ktype kof kthe kblood kon kthe kunderwear
□Interrogate k the k father
□Isolate kthe kparents kand kchild
□Determine kvon kWillebrandkfactor klevels
, □Measure kfibronectinkinkthekvagina
description kConsideration kof kfactitious kdisorder kby kproxy kshould kbe ktriggered kwhen kthe kreported ksymptoms kare
krepeatedly k notedkbykonlykonekparent,kappropriatektestingkfailsktokconfirmkakdiagnosis,kandkseeminglykappropriate
ktreatmentkiskineffective.
Atktimes,kthekchild'sksymptoms,ktheirkcourse,korkthekresponsektoktreatmentkmaykbekincompatiblekwithkanykrecognizedkdisease.
k Preverbalkchildrenkarekusuallykinvolved.kBleedingkiskakparticularlykcommonkpresentation.kThiskmaykbekcausedkbykaddingkdyes
k k to ksamples, kadding kblood k(e.g., kfrom kthe kmother) kto kthe kchild's ksample, kor kgiving kthe kchild kan kanticoagulant k(e.g.,
kwarfarin). k (See kChapter k37, kpage k146.)
3. Munchausen ksyndrome kby kproxy kis kcharacterized kby kall kof k the kfollowing kEXCEPT:
□Motherkwho kappears kdevoted kand kwins koverkmembers kofkcarekteam
□Multiple k hospitalizations k and k investigations k without k diagnosis
□Symptomskonkhistorykbutknotkwitnessedkbykmedicalkteam
■ Symptomskoccurringkinkpresencekofkdifferentkcaregiversk(e.g.,kwhilekmotherkiskoutkofktown)
□Use kof kmedications kor ktoxins
description kSymptoms kin kyoung kchildren kare kmostly kassociated kwith kproximity kof kthe koffending kcaregiver kto kthe kchild.
kThe k mother kmay kpresent kas ka kdevoted kor keven kmodel kparent kwho kforms kclose krelationships kwith kmembers kof kthe
khealth kcare k team. kWhile kappearing kvery kinterested kin kher kchild's kcondition, kshe kmay kbe krelatively kdistant
kemotionally. k(See kChapter k37, k kpage k146.)
4. Which kstatement k is kfalse?
■ Malnutrition kis kthe ksecond kleading kcause kof kacquired kimmune kdeficiency kworldwide kbehind kHIV kinfection
□Zinc kis kimportant kinkimmunekfunction kand klinear kgrowth
□Kwashiorkor kand kmarasmus kare krare kin kdeveloped kcountries
□The kWestern kdiet kis kassociated kwith kincreased knoncommunicable kdisease
descriptionkTheksignificantkglobalkburdenkofkmalnutritionkandkundernutritionkiskthekleadingkworldwidekcausekofkacquired
k immunodeficiencykandkthekmajorkunderlyingkfactorkforkmorbiditykandkmortalitykgloballykforkchildrenk<5kyrkofkage.kZinckis
ka k micronutrientkthatksupportskmultiplekmetabolickfunctionskinkthekbody,kiskessentialkforknormalkimmunekfunctioning,kandkis
k requiredktoksupportklinearkgrowth;kzinckdeficiencykiskassociatedkwithkimpairedkimmunekfunctioningkandkpoorklinearkgrowth.
kIn k parallelktokthekriskkforknutrientkandkenergykdeficiencies,kissueskrelatingktokexcesseskposekimportantkchallengeskbecause
kofktheir k negative khealth keffects, ksuch kas kobesity kor kcardiovascular kdisease krisk kfactors. kThe knutrition ktransition
kunder kway kin kthe
, developing kworld kfrom ktraditional kdiets kto kthe kWestern kdiet khas kbeen kassociated kwith kincreases kin knoncommunicable
k diseases, koften kcoexisting kwith kundernutrition kand kmalnutrition, kobserved ksometimes kin kthe ksame kcommunities kor keven kthe
k same kfamilies. k(See ke41-1.)
5. Components k of k energy k expenditure k in kchildren k include:
□Thermalkeffectkofkfood
□Basal kmetabolic k rate
□Energy kfor kphysical kactivity
□Energyktoksupportkgrowth
■ All kof kthe kabove
description kThe k3 kcomponents kof kenergy kexpenditure kin kadults kare kthe kbasal kmetabolic krate, kthe kthermal keffect
kof kfood k (energy krequired kfor kdigestion kand kabsorption), kand kenergy kfor kphysical kactivity. kAdditional kenergy kintake
kand kexpenditure k are krequired kto ksupport kgrowth kand kdevelopment kfor kchildren. k(See ke41-4.)
6. Which kof kthe kfollowing kclinical kscenarios kincreases kthe krisk kof kvitamin kA kdeficiency?
□Vegetarian k diet
□Chronic k intestinal k disorders
□Zinc kdeficiency
■ B kand kC
□Allkofkthekabove
description kVitamin kA kis kan kessential kmicronutrient kbecause kit kcannot kbe kbiogenerated kde knovo kby kanimals. kIt
kmust kbe k obtained kfrom kplants kin kthe kform kof kprovitamin-A kcarotenoids. kIn kthe kUSA, kgrains kand kvegetables ksupply
kapproximately k 55% kand kdairy kand kmeat kproducts ksupply kapproximately k30% kof kvitamin kA kintake kfrom kfood.
kVitamin kA kand kthe k provitamins-Akarekfatksoluble,kandktheirkabsorptionkdependskonkthekpresencekofkadequateklipidkand
kproteinkwithinkthekmeal.
Chronic kintestinal kdisorders kor klipid kmalabsorption ksyndromes kcan kresult kin kvitamin kA kdeficiency. kIn kdeveloping kcountries,
k subclinicalkorkclinicalkzinckdeficiencykcankincreasekthekriskkofkvitaminkAkdeficiency.kTherekiskalsoksomekevidencekofkmarginal
k zinc kintakes k in kchildren kin kthe kUSA. k(See kChapter k45, k page k188.)
7. Which kstatement k about k vitamin k A k toxicity k is k NOT k true?
