Test Bank For Rubin's Pathology: Clinicopathologic Foundations of Medicine 7th Edition by Strayer ISBN:9781451183900, All 34 Chapters Covered, Verified Latest Edition

TESTBANK i




RUBIN'S PATHOLOGY: CLINICOPATHOLOGIC FOUNDATIONS OF
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MEDICINE 7TH EDITION
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DAVID S. STRAYER, EMANUEL RUBIN
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,Test iBank iRubin's iPathology: iClinicopathologic iFoundations iof iMedicine i7th iEdition

Table iof iContents:
Chapter i1: iCell iAdaptation, iInjury iand iDeath
Chapter i2: iInflammation
Chapter i3: iRepair, iRegeneration iand iFibrosis
Chapter i4: iImmunopathology
Chapter i5: iNeoplasia
Chapter i6: iDevelopmental iand iGenetic iDiseases
Chapter i7: iHemodynamic iDisorders
Chapter i8: iEnvironmental iand iNutritional iPathology
Chapter i9: iInfectious iand iParasitic iDiseases
Section iII: iPathogenesis iof iSystemic iConditions iExpandable isection
Chapter i10: iAging
Chapter i11: iSystemic iAutoimmune iDiseases
Chapter i12: iSepsis
Chapter i13: iObesity iand iDiabetes iMellitus
Chapter i14: iThe iPathology iof iPregnancy
Chapter i15: iThe iAmyloidoses
Section iIII: iDiseases iof iIndividual iOrgan iSystemsExpandable isection
Chapter i16: iBlood iVessels
Chapter i17: iThe iHeart
Chapter i18: iThe iRespiratory iSystem
Chapter i19: iThe iGastrointestinal iTract
Chapter i20: iThe iLiver iand iBiliary iSystem
Chapter i21: iThe iPancreas
Chapter i22: iThe iKidney
Chapter i23: iThe iLower iUrinary iTract iand iMale iReproductive iSystem
Chapter i24: iThe iFemale iReproductive iSystem iand iPeritoneum
Chapter i25: iThe iBreast
Chapter i26: iHematopathology
Chapter i27: iThe iEndocrine iSystem
Chapter i28: iThe iSkin
Chapter i29: iThe iHead iand iNeck
Chapter i30: iBones, iJoints iand iSoft iTissue
Chapter i31: iSkeletal iMuscle iand iPeripheral iNervous iSystem
Chapter i32: iThe iCentral iNervous iSystem
Chapter i33: iThe iEye
Chapter i34: iForensic iPathology

,Rubin's iPathology: iClinicopathologic iFoundations iof
iMedicineChapter i1: iCell iAdaptation, iInjury iand iDeath




Ischemia iand iother itoxic iinjuries iincrease ithe iaccumulation iof iintracellular icalcium ias ia iresult
1. of:
A) release iof istored icalcium ifrom ithe imitochondria.
B) improved iintracellular ivolume iregulation.
C) decreased iinflux iacross ithe icell imembrane.
D) attraction iof icalcium ito ifatty iinfiltrates.
The ipatient iis ifound ito ihave iliver idisease, iresulting iin ithe iremoval iof ia ilobe iof ihis iliver.
2. Adaptation ito ithe ireduced isize iof ithe iliver ileads ito i_ of ithe iremaining iliver icells.
A) metaplasia
B) organ iatrophy
C) compensatory ihyperplasia
D) physiologic ihypertrophy
A iperson ieating ipeanuts istarts ichoking iand icollapses. iHis iairway iobstruction iis ipartially
icleared, ibut ihe iremains ihypoxic iuntil ihe ireaches ithe ihospital. iThe iprolonged icell ihypoxia

3. caused ia icerebral iinfarction iand iresulting _ iin ithe ibrain.
A) caspase iactivation
B) coagulation inecrosis
C) rapid iphagocytosis
D) protein ip53 ideficiency
Bacteria iand iviruses icause icell idamage iby , iwhich iis iunique ifrom ithe iintracellular
4. damage icaused iby iother iinjurious iagents.
A) disrupting ithe isodium/potassium iATPase ipump
B) interrupting ioxidative imetabolism iprocesses
C) replicating iand iproducing icontinued iinjury
D) decreasing iprotein isynthesis iand ifunction
The ipatient ihas ia iprolonged iinterruption iin iarterial iblood iflow ito ihis ileft ikidney, icausing
5. hypoxic icell iinjury iand ithe irelease iof ifree iradicals. iFree iradicals idamage icells iby:
A) destroying iphospholipids iin ithe icell imembrane.
B) altering ithe iimmune iresponse iof ithe icell.
C) disrupting icalcium istorage iin ithe icell.
D) inactivation iof ienzymes iand imitochondria.

, 6. Injured icells ihave iimpaired iflow iof isubstances ithrough ithe icell imembrane ias ia iresult iof:
A) increased ifat iload.
B) altered ipermeability.
C) altered iglucose iutilization.
D) increased isurface ireceptors.
7. Reversible iadaptive iintracellular iresponses iare iinitiated iby:
A) stimulus ioverload.
B) genetic imutations.
C) chemical imessengers.
D) mitochondrial iDNA.
8. Injured icells ibecome ivery iswollen ias ia iresult iof:
A) increased icell iprotein isynthesis.
B) altered icell ivolume iregulation.
C) passive ientry iof ipotassium iinto ithe icell.
D) bleb iformation iin ithe iplasma imembrane.
A idiabetic ipatient ihas iimpaired isensation, icirculation, iand ioxygenation iof ihis ifeet. iHe isteps
ion ia ipiece iof iglass, ithe iwound idoes inot iheal, iand ithe iarea itissue ibecomes inecrotic. iThe

inecrotic

9. cell ideath iis icharacterized iby:
A) rapid iapoptosis.
B) cellular irupture.
C) shrinkage iand icollapse.
D) chronic iinflammation.
A i99-year-old iwoman ihas iexperienced ithe idecline iof icell ifunction iassociated iwith iage. iA
10. group iof itheories i of icellular iaging ifocus ion iprogrammed:
A) changes iwith igenetic iinfluences.
B) elimination iof icell ireceptor isites.
C) insufficient itelomerase ienzyme.
D) DNA imutation ior ifaulty irepair.
An i89-year-old ifemale ipatient ihas iexperienced isignificant idecreases iin iher imobility iand
istamina iduring ia i3-week ihospital istay ifor ithe itreatment iof ia ifemoral ihead ifracture. iWhich iof

ithe ifollowing iphenomena imost ilikely iaccounts ifor ithe ipatients idecrease iin imuscle ifunction

11. that iunderlies iher ireduced imobility?
A) Impaired imuscle icell imetabolism iresulting ifrom imetaplasia
B) Dysplasia ias ia iconsequence iof iinflammation iduring ibone iremodeling
C) Disuse iatrophy iof imuscle icells iduring ia iprolonged iperiod iof iimmobility