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Examen

NSG223 Exam 2 Study Guide 2026/2027 | Practice Questions & Answers

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Prepare for NSG223 Exam 2 with this 2026/2027 study guide. Includes practice questions, detailed answers, and strategies to help nursing students master core concepts, clinical decision-making, and patient care principles for exam success.

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NSG223
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NSG223

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Subido en
23 de enero de 2026
Número de páginas
9
Escrito en
2025/2026
Tipo
Examen
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Practice questions for this set


Learn 1 /7 Study using Learn




headache, confusion, drowsiness, ⬆RR and depth, nausea, and vomiting


Peripheral vasodilation and ⬇cardiac output when pH<7


⬇BP, cold and clammy skin, dysrhythmias, and shock


Chronic with CKD



Select the correct term



Symptoms related to dehydration and
1 Signs and symptoms of metabolic acidosis 2
electrolyte imbalance



3 Indicators of respiratory failure and hypoxia 4 Signs of chronic kidney disease progression



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Terms in this set (117)




NSG223 - Exam 2 Study Guide NSG223 - Exam 2 Study Guide ?? Verified questions & answers with solution

, NSG223 - Exam 2 Study Guide ?? Verified questionsNSG223
& answers
- Exam
with2solution.
Study Guide.pdf
Page 2 of 9 ?? Verified questions & answers with solution


headache, confusion, drowsiness, ⬆RR and depth, nausea, and vomiting


Peripheral vasodilation and ⬇cardiac output when pH<7
Signs and symptoms of metabolic
acidosis
⬇BP, cold and clammy skin, dysrhythmias, and shock


Chronic with CKD

What is the cardinal feature of metabolic A decrease in the serum bicarbonate level
acidosis?

Hyperventilation, with a resulting Metabolic acidosis
decrease in PaCO2, is an expected
compensatory reaction to which acid--
base disorder?

What is the most common blood-borne Hepatitis C
disease in the United States, and is the
leading cause of liver cancer in many
countries?

What could be considered the least Hepatitis A
dangerous of the hepatitis infections,
causes abdominal pain, jaundice, nausea,
and vomiting that can occasionally last for
months?

Main prevention methods for hepatitis strict adherence to infection control (Standard Precautions and Safe Injection
infections? Practices)

Pathophysiology of PE blood clot, thrombus, emboli (air, fat, amniotic fluid, septic [bacterial]), or A-fib

What happens when a thrombus Alveolar dead space is increased which leads to impaired gas exchange
completely or partially obstructs a
pulmonary artery or its branches?

What results in an increase in pulmonary Substances released from the clot and surrounding area cause blood vessels and
vascular resistance—a reaction that bronchioles to constrict
compounds the V./Q. imbalance?

Depends on size of thrombus and area of pulmonary artery occluded by the
thrombus


Dyspnea (most frequent symptom)
Clinical manifestations of PE
Chest pain (sudden)
Anxiety, fever, tachycardia, apprehension, cough, diaphoresis, hemoptysis, and
syncope
Tachypnea (most frequent sign)

Obstruction of the pulmonary artery Dyspnea, sudden substernal pain, rapid and weak pulse, shock, syncope, and
results in? sudden death.

Active leg exercises to avoid venous stasis, early ambulation, anti-embolism
Prevention of PE stockings, compression devices, feet rest on floor/chair (dependent position, not
dangled). Avoid prolonged use of IV catheters and crossing legs.

Treatment goal of PE To dissolve (lyse) the existing emboli and prevent new ones from forming

Improve respiratory and vascular status, anticoagulation therapy, thrombolytic
Treatment of PE
therapy, and surgical intervention

Nasal oxygen, IV infusion lines, vasopressor therapy for hypotension unrelieved
Emergency management of PE by IV fluids, ECG for dysrhythmias, blood draw (CBC, electrolytes, coagulation),
Foley catheter, IV morphine/sedatives

Embolectomy
Surgical management of PE
Inferior vena cava (IVC) filtration

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